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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Schema
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We began analyzing https://link.springer.com/article/10.1007/s10555-010-9223-6, but it redirected us to https://link.springer.com/article/10.1007/s10555-010-9223-6. The analysis below is for the second page.

Title[redir]:
Interactions between lymphocytes and myeloid cells regulate pro- versus anti-tumor immunity | Cancer and Metastasis Reviews
Description:
Tumor-associated myeloid cells have been implicated in regulating many of the “hallmarks of cancer” and thus fostering solid tumor development and metastasis. However, the same innate leukocytes also participate in anti-tumor immunity and restraint of malignant disease. While many factors regulate the propensity of myeloid cells to promote or repress cancerous growths, polarized adaptive immune responses by B and T lymphocytes have been identified as regulators of many aspects of myeloid cell biology by specifically regulating their functional capabilities. Here, we detail the diversity of heterogeneous B and T lymphocyte populations and their impacts on solid tumor development through their abilities to regulate myeloid cell function in solid tumors.

Matching Content Categories {📚}

  • Science
  • Education
  • Health & Fitness

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🏙️ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 96,105,781 visitors per month in the current month.

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How Does Doi.org Make Money? {💸}

The income method remains a mystery to us.

While profit motivates many websites, others exist to inspire, entertain, or provide valuable resources. Websites have a variety of goals. And this might be one of them. Doi.org might be making money, but it's not detectable how they're doing it.

Keywords {🔍}

cells, google, scholar, article, cas, pubmed, cancer, cell, tumor, immune, development, myeloid, responses, macrophages, immunity, lymphocytes, antitumor, activation, immunology, journal, role, metastasis, regulatory, nature, regulate, programs, angiogenesis, human, growth, cytokines, production, carcinoma, research, coussens, tumors, reviews, inflammation, including, solid, lymphocyte, macrophage, progression, treg, clinical, carcinogenesis, protumor, states, expression, versus, turn,

Topics {✒️}

tumor-infiltrating foxp3-positive regulatory cell/immunoglobulin/fcγr signaling axis tumor-infiltrating foxp3-cd4+cd25+ cell-mediated antigen-specific immunotherapy regulate anti-tumor programs anti-tumor immune responses anti-tumor immune programs enhance anti-tumor responses favor pro-tumor n2 article download pdf tumor-immune surveillance programs neutralize m2-type macrophage pro-tumor myeloid cells pro-inflammatory cytokines il1 promoting cxcr-2-dependent angiogenesis regulating pro-tumor properties immune-competent mouse models t-helper-cell differentiation strong antigen-specific activation cell-dependent tumor immunity similar pro-tumor role tumor-specific th17 cells transforming growth factor il17-producing cells correlates turn activates ikk-α tumor necrosis factor lymphocyte-mediated chronic inflammation interleukin-17 promotes angiogenesis anti-tumor programs ifnγ-producing th1 cells pro-tumor pathways cd4+cd25+foxp3+ regulatory tumor-bearing state accessory-cell-dependent activation anti-tumor programming human cd4+ il-17-producing privacy choices/manage cookies anti-tumor immunity anti-tumor antibodies increased tumor size enhanced humoral responses alter adaptive immunity immune-suppressive programs [25] pro-tumor immunity myeloid suppressor cells anti-tumor activities fostered immune surveillance anti-tumor ctl multiple immunosuppressive programs prostate cancer cells

Schema {🗺️}

WebPage:
      mainEntity:
         headline:Interactions between lymphocytes and myeloid cells regulate pro- versus anti-tumor immunity
         description:Tumor-associated myeloid cells have been implicated in regulating many of the “hallmarks of cancer” and thus fostering solid tumor development and metastasis. However, the same innate leukocytes also participate in anti-tumor immunity and restraint of malignant disease. While many factors regulate the propensity of myeloid cells to promote or repress cancerous growths, polarized adaptive immune responses by B and T lymphocytes have been identified as regulators of many aspects of myeloid cell biology by specifically regulating their functional capabilities. Here, we detail the diversity of heterogeneous B and T lymphocyte populations and their impacts on solid tumor development through their abilities to regulate myeloid cell function in solid tumors.
         datePublished:2010-04-21T00:00:00Z
         dateModified:2010-04-21T00:00:00Z
         pageStart:309
         pageEnd:316
         license:https://creativecommons.org/licenses/by-nc/2.0
         sameAs:https://doi.org/10.1007/s10555-010-9223-6
         keywords:
            Cancer
            Inflammation
            Lymphocyte
            Macrophage
            Metastasis
            Cancer Research
            Oncology
            Biomedicine
            general
         image:
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         author:
               name:David G. DeNardo
               affiliation:
                     name:University of California
                     address:
                        name:Department of Pathology, University of California, San Francisco, USA
                        type:PostalAddress
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               name:Pauline Andreu
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                     name:University of California
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                        name:Department of Pathology, University of California, San Francisco, USA
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                        name:Department of Pathology, University of California, San Francisco, USA
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                     address:
                        name:Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, USA
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ScholarlyArticle:
      headline:Interactions between lymphocytes and myeloid cells regulate pro- versus anti-tumor immunity
      description:Tumor-associated myeloid cells have been implicated in regulating many of the “hallmarks of cancer” and thus fostering solid tumor development and metastasis. However, the same innate leukocytes also participate in anti-tumor immunity and restraint of malignant disease. While many factors regulate the propensity of myeloid cells to promote or repress cancerous growths, polarized adaptive immune responses by B and T lymphocytes have been identified as regulators of many aspects of myeloid cell biology by specifically regulating their functional capabilities. Here, we detail the diversity of heterogeneous B and T lymphocyte populations and their impacts on solid tumor development through their abilities to regulate myeloid cell function in solid tumors.
      datePublished:2010-04-21T00:00:00Z
      dateModified:2010-04-21T00:00:00Z
      pageStart:309
      pageEnd:316
      license:https://creativecommons.org/licenses/by-nc/2.0
      sameAs:https://doi.org/10.1007/s10555-010-9223-6
      keywords:
         Cancer
         Inflammation
         Lymphocyte
         Macrophage
         Metastasis
         Cancer Research
         Oncology
         Biomedicine
         general
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                     type:PostalAddress
                  type:Organization
            type:Person
            name:Pauline Andreu
            affiliation:
                  name:University of California
                  address:
                     name:Department of Pathology, University of California, San Francisco, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Lisa M. Coussens
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                  name:University of California
                  address:
                     name:Department of Pathology, University of California, San Francisco, USA
                     type:PostalAddress
                  type:Organization
                  name:University of California
                  address:
                     name:Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, USA
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      name:University of California
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         name:Department of Pathology, University of California, San Francisco, USA
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            name:University of California
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               name:Department of Pathology, University of California, San Francisco, USA
               type:PostalAddress
            type:Organization
      name:Lisa M. Coussens
      affiliation:
            name:University of California
            address:
               name:Department of Pathology, University of California, San Francisco, USA
               type:PostalAddress
            type:Organization
            name:University of California
            address:
               name:Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, USA
               type:PostalAddress
            type:Organization
      email:[email protected]
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      name:Department of Pathology, University of California, San Francisco, USA
      name:Department of Pathology, University of California, San Francisco, USA
      name:Department of Pathology, University of California, San Francisco, USA
      name:Helen Diller Family Comprehensive Cancer Center, University of California, San Francisco, USA

External Links {🔗}(437)

Analytics and Tracking {📊}

  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • Prism.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {📦}

  • Crossref

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