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LINK . SPRINGER . COM {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Link.springer.com Make Money
  6. Keywords
  7. Topics
  8. Schema
  9. External Links
  10. Analytics And Tracking
  11. Libraries

We are analyzing https://link.springer.com/chapter/10.1007/978-0-387-34814-8_14.

Title:
CD4+ T Cells Cooperate With Macrophages for Specific Elimination of MHC Class II-Negative Cancer Cells | SpringerLink
Description:
Our present knowledge of how T cells eliminate cancer is mainly based on memory immune responses investigated with vaccinated mice1,2. In-vivo depletion studies with anti-CD4 monoclonal antibodies (mAb) have revealed that the antitumor immunity conferred by...
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {πŸ“š}

  • Education
  • Science
  • Health & Fitness

Content Management System {πŸ“}

What CMS is link.springer.com built with?

Custom-built

No common CMS systems were detected on Link.springer.com, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of link.springer.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 8,149,968 visitors per month in the current month.

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How Does Link.springer.com Make Money? {πŸ’Έ}

We can't figure out the monetization strategy.

Not all websites focus on profit; some are designed to educate, connect people, or share useful tools. People create websites for numerous reasons. And this could be one such example. Link.springer.com could have a money-making trick up its sleeve, but it's undetectable for now.

Keywords {πŸ”}

google, scholar, pubmed, cas, article, cells, tumor, immunity, cancer, cell, exp, med, usa, macrophages, immunol, immune, vivo, bogen, specific, class, proc, natl, acad, sci, role, antigen, tumour, privacy, cookies, content, information, publish, paper, corthay, antitumor, download, res, dembic, schreiber, surveillance, nature, springer, search, innate, adaptive, mhc, iinegative, mice, antibodies, lymphoma,

Topics {βœ’οΈ}

t-cell receptor-transgenic mice long-lasting anti-tumor immunity anti-class ii antibodies macrophage-activating factor produced antigen-specific cd4+ t-cell rikshospitalet-radiumhospitalet medical center month download article/chapter cell-dependent tumor rejection tumor-specific transplantation antigens tumor-capturing dendritic cells conference paper crossroads myelomas activate tumor-specific anti-cd4 monoclonal antibodies cell-dependent tumor eradication mhc class cells eliminate cancer naive idiotype-specific cd4+ murine tumor expressing privacy choices/manage cookies b-cell tumors tumor cell responses perforin-mediated cytotoxicity tumor-specific antigen model tumor antigen disseminated murine leukemia paper cite device instant download antitumor immune response tumor-specific lyt-1+2 anti-idiotypic antibodies antitumor immunity conferred differential tumor surveillance eradicating tumor cells google scholar shape tumour immunogenicity immune lymphoid cells cell clones specific single cell level immunologically specific killing european economic area eradicate pulmonary metastases epithelial malignancies effected basement membrane complexes download preview pdf dendritic cells purified phagocytic dendritic cells th2 cell clones helper cell requirement conditions privacy policy idiotype-specific cd4+

Schema {πŸ—ΊοΈ}

ScholarlyArticle:
      headline:CD4+ T Cells Cooperate With Macrophages for Specific Elimination of MHC Class II-Negative Cancer Cells
      pageEnd:208
      pageStart:195
      image:https://media.springernature.com/w153/springer-static/cover/book/978-0-387-34814-8.jpg
      genre:
         Biomedical and Life Sciences
         Biomedical and Life Sciences (R0)
      isPartOf:
         name:Crossroads between Innate and Adaptive Immunity
         isbn:
            978-0-387-34814-8
            978-0-387-34813-1
         type:Book
      publisher:
         name:Springer US
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Alexandre Corthay
            affiliation:
                  name:University of Oslo and Rikshospitalet-Radiumhospitalet Medical Center
                  address:
                     name:Institute of Immunology, University of Oslo and Rikshospitalet-Radiumhospitalet Medical Center, Oslo, Norway
                     type:PostalAddress
                  type:Organization
            type:Person
      keywords:Myeloma Cell, SCID Mouse, Antitumor Immunity, Tumor Rejection, Tumor Eradication
      description:Our present knowledge of how T cells eliminate cancer is mainly based on memory immune responses investigated with vaccinated mice1,2. In-vivo depletion studies with anti-CD4 monoclonal antibodies (mAb) have revealed that the antitumor immunity conferred by prophylactic vaccination is usually CD4+ T cell dependent. CD4+ T cells were required for vaccination-induced immunity against the B16 melanoma3–5, against the Mc51.9 fibrosarcoma6, against the J558 plasmacytoma7, and against the A20 lymphoma7.
      datePublished:2007
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
      context:https://schema.org
Book:
      name:Crossroads between Innate and Adaptive Immunity
      isbn:
         978-0-387-34814-8
         978-0-387-34813-1
Organization:
      name:Springer US
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:University of Oslo and Rikshospitalet-Radiumhospitalet Medical Center
      address:
         name:Institute of Immunology, University of Oslo and Rikshospitalet-Radiumhospitalet Medical Center, Oslo, Norway
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:Alexandre Corthay
      affiliation:
            name:University of Oslo and Rikshospitalet-Radiumhospitalet Medical Center
            address:
               name:Institute of Immunology, University of Oslo and Rikshospitalet-Radiumhospitalet Medical Center, Oslo, Norway
               type:PostalAddress
            type:Organization
PostalAddress:
      name:Institute of Immunology, University of Oslo and Rikshospitalet-Radiumhospitalet Medical Center, Oslo, Norway
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

External Links {πŸ”—}(154)

Analytics and Tracking {πŸ“Š}

  • Google Tag Manager

Libraries {πŸ“š}

  • Clipboard.js

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