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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
  10. External Links
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We began analyzing https://link.springer.com/article/10.1007/s00262-011-1139-7, but it redirected us to https://link.springer.com/article/10.1007/s00262-011-1139-7. The analysis below is for the second page.

Title[redir]:
MicroRNA regulation in cancer-associated fibroblasts | Cancer Immunology, Immunotherapy
Description:
The microenvironment of cancer cells has proven to be a critical component of tumors that strongly influences cancer development and progression into invasive and metastatic disease. Compared to normal tissue, dramatic differences in gene expression occur in multiple cell types that constitute the tumor microenvironment including cancer-associated fibroblasts (CAFs) that are important stromal components of growing tumors. In this review, we present recent advances in understanding how microRNAs are deregulated in cancer-associated fibroblasts (CAFs) and how this affects tumor biology. The microRNA signature of CAFs is discussed with respect to their functional relevance to tumor cells as well as other cell types involved in tumor homeostasis.

Matching Content Categories {πŸ“š}

  • Education
  • Health & Fitness
  • Science

Content Management System {πŸ“}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of doi.org audience?

πŸ™οΈ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 98,426,998 visitors per month in the current month.

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How Does Doi.org Make Money? {πŸ’Έ}

We're unsure if the website is profiting.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Doi.org might be plotting its profit, but the way they're doing it isn't detectable yet.

Keywords {πŸ”}

article, google, scholar, pubmed, cancer, cas, fibroblasts, tumor, cell, cells, microrna, cancerassociated, breast, microenvironment, res, research, stromal, stroma, content, access, nat, polyak, human, biol, privacy, cookies, aprelikova, green, metastatic, normal, expression, micrornas, myofibroblasts, wang, nature, information, publish, search, review, biology, role, rev, genet, growth, richardson, epithelial, prostate, hanahan, dev, tumorigenesis,

Topics {βœ’οΈ}

col-i-enriched fibrotic environment month download article/chapter bone-marrow-derived myofibroblasts contribute nf-kappab-dependent manner orchestrate tumor-promoting inflammation elevated sdf-1/cxcl12 secretion nf-kappab-dependent induction tlr-triggered dendritic cells article cancer immunology full article pdf cancer-induced stromal reaction privacy choices/manage cookies bone marrow contribution focussed research reviews human cancer metastasis affects tumor biology related subjects metastasis-related micrornas independent genetic alterations hypoxia-mediated selection intramural research program microenvironment cross-talk breast cancers article aprelikova primary breast cancer tumor cell proliferation tumour-stroma interaction epithelium reveals multiplicity frequent somatic mutations innate immune responses breast cancer stroma dormant cells induced gene expression occur normal prostate fibroblasts european economic area present recent advances suppress neoplastic development normal colonic mucosa cantemir-stone cz tlsty td da silva sd mutual paracrine effects rate-limiting determinant deliver small rna mar-binding protein organizations imply endorsement pleiotropically acting microrna microrna 148a production important stromal components anti-cancer therapy

Questions {❓}

  • Micke P, Ostman A (2004) Tumour-stroma interaction: cancer-associated fibroblasts as novel targets in anti-cancer therapy?
  • Radisky DC, Kenny PA, Bissell MJ (2007) Fibrosis and cancer: do myofibroblasts come also from epithelial cells via EMT?
  • Rubin H (2008) Contact interactions between cells that suppress neoplastic development: can they also explain metastatic dormancy?

Schema {πŸ—ΊοΈ}

WebPage:
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         headline:MicroRNA regulation in cancer-associated fibroblasts
         description:The microenvironment of cancer cells has proven to be a critical component of tumors that strongly influences cancer development and progression into invasive and metastatic disease. Compared to normal tissue, dramatic differences in gene expression occur in multiple cell types that constitute the tumor microenvironment including cancer-associated fibroblasts (CAFs) that are important stromal components of growing tumors. In this review, we present recent advances in understanding how microRNAs are deregulated in cancer-associated fibroblasts (CAFs) and how this affects tumor biology. The microRNA signature of CAFs is discussed with respect to their functional relevance to tumor cells as well as other cell types involved in tumor homeostasis.
         datePublished:2011-11-16T00:00:00Z
         dateModified:2011-11-16T00:00:00Z
         pageStart:231
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            Immunology
            Cancer Research
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      headline:MicroRNA regulation in cancer-associated fibroblasts
      description:The microenvironment of cancer cells has proven to be a critical component of tumors that strongly influences cancer development and progression into invasive and metastatic disease. Compared to normal tissue, dramatic differences in gene expression occur in multiple cell types that constitute the tumor microenvironment including cancer-associated fibroblasts (CAFs) that are important stromal components of growing tumors. In this review, we present recent advances in understanding how microRNAs are deregulated in cancer-associated fibroblasts (CAFs) and how this affects tumor biology. The microRNA signature of CAFs is discussed with respect to their functional relevance to tumor cells as well as other cell types involved in tumor homeostasis.
      datePublished:2011-11-16T00:00:00Z
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         Cancer Research
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External Links {πŸ”—}(237)

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