DOI . ORG {
}
We began analyzing https://link.springer.com/chapter/10.1007/978-94-007-2199-9_6, but it redirected us to https://link.springer.com/chapter/10.1007/978-94-007-2199-9_6. The analysis below is for the second page.
Title[redir]:
Vitamin C Transport and Its Role in the Central Nervous System | SpringerLink
Description:
Vitamin C, or ascorbic acid, is important as an antioxidant and participates in numerous cellular functions. Although it circulates in plasma in micromolar concentrations, it reaches millimolar concentrations in most tissues. These high ascorbate cellular...
Title[redir]:
Vitamin C Transport and Its Role in the Central Nervous System | SpringerLink
Description:
Vitamin C, or ascorbic acid, is important as an antioxidant and participates in numerous cellular functions. Although it circulates in plasma in micromolar concentrations, it reaches millimolar concentrations in most tissues. These high ascorbate cellular...
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Keywords {🔍}
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Topics {✒️}
month download article/chapter high-pressure liquid chromatography liquid chromatography/mass spectrometry na+-dependent active transport anoxia-tolerant reptiles contrasted mc3t3-e1 osteoblastic cells dehydro-l-ascorbic acid nadh-ascorbate free radical nadph-dependent dehydroascorbate reductase high-affinity sodium-vitamin systemic kainate-induced neurotoxicity rat blood-retinal barrier water-soluble vitamins water soluble vitamins sh-sy5y neuroblastoma cells ascorbic-acid transporter slc23a1 glutathione-dependent dehydroascorbate reductase dehydroascorbic acid-mediated reduction ischemia-reperfusion injury models human sodium-dependent vitamin privacy choices/manage cookies l-ascorbic acid central nervous system device instant download dehydroascorbic acid reductase human endothelial cells sodium-dependent vitamin nitric oxide increased extracellular levels dehydroascorbic acid measurements dehydroascorbic acid solutions blood-brain barrier intracellular ascorbic acid free radical production ascorbyl free radical physiologically induced release ascorbic acid homeostasis van mil se outer mitochondrial membrane high physiological concentrations anoxia-intolerant species van der zee widespread cerebral hemorrhage focal cerebral ischaemia ascorbate free radical ascorbic acid transporter hypothalamic glial cells cerebellar granule neurons sodium-coupled vitamin selenoenzyme thioredoxin reductase
Schema {🗺️}
ScholarlyArticle:
headline:Vitamin C Transport and Its Role in the Central Nervous System
pageEnd:103
pageStart:85
image:https://media.springernature.com/w153/springer-static/cover/book/978-94-007-2199-9.jpg
genre:
Biomedical and Life Sciences
Biomedical and Life Sciences (R0)
isPartOf:
name:Water Soluble Vitamins
isbn:
978-94-007-2199-9
978-94-007-2198-2
type:Book
publisher:
name:Springer Netherlands
logo:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
type:ImageObject
type:Organization
author:
name:James M. May
affiliation:
name:Vanderbilt University School of Medicine
address:
name:Departments of Medicine and Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, USA
type:PostalAddress
type:Organization
email:[email protected]
type:Person
keywords:Brain, SVCT2, Ascorbate transport, Dehydroascorbate, Neurons, Glutamate
description:Vitamin C, or ascorbic acid, is important as an antioxidant and participates in numerous cellular functions. Although it circulates in plasma in micromolar concentrations, it reaches millimolar concentrations in most tissues. These high ascorbate cellular concentrations are thought to be generated and maintained by the SVCT2 (Slc23a2), a specific transporter for ascorbate. The vitamin is also readily recycled from its oxidized forms inside cells. Neurons in the central nervous system (CNS) contain some of the highest ascorbic acid concentrations of mammalian tissues. Intracellular ascorbate serves several functions in the CNS, including antioxidant protection, peptide amidation, myelin formation, synaptic potentiation, and protection against glutamate toxicity. The importance of the SVCT2 for CNS function is supported by the finding that its targeted deletion in mice causes widespread cerebral hemorrhage and death on post-natal day 1. Neuronal ascorbate content as maintained by this protein also has relevance for human disease, since ascorbate supplements decrease infarct size in ischemia-reperfusion injury models of stroke, and since ascorbate may protect neurons from the oxidant damage associated with neurodegenerative diseases such as Alzheimer’s, Parkinson’s, and Huntington’s. The aim of this review is to assess the role of the SVCT2 in regulating neuronal ascorbate homeostasis and the extent to which ascorbate affects brain function and antioxidant defenses in the CNS.
datePublished:2012
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