DOI . ORG {
}
We began analyzing https://link.springer.com/chapter/10.1007/978-3-540-73677-6_8, but it redirected us to https://link.springer.com/chapter/10.1007/978-3-540-73677-6_8. The analysis below is for the second page.
Title[redir]:
Remyelination in Experimental Models of Toxin-Induced Demyelination | SpringerLink
Description:
Remyelination is the regenerative process by which demyelinated axons are reinvested with new myelin sheaths. It is associated with functional recovery and maintenance of axonal health. It occurs as a spontaneous regenerative response following demyelination in a...
Title[redir]:
Remyelination in Experimental Models of Toxin-Induced Demyelination | SpringerLink
Description:
Remyelination is the regenerative process by which demyelinated axons are reinvested with new myelin sheaths. It is associated with functional recovery and maintenance of axonal health. It occurs as a spontaneous regenerative response following demyelination in a...
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Keywords {🔍}
google, scholar, pubmed, article, cas, remyelination, demyelination, blakemore, cns, franklin, oligodendrocyte, neurosci, cells, rjm, brain, cell, adult, spinal, cord, central, rat, neurol, system, multiple, sclerosis, experimental, neuropathol, nervous, chapter, lesions, demyelinated, transplantation, glia, neurobiol, zhao, progenitor, study, induced, oligodendrocytes, demyelinating, models, appl, gilson, crang, glial, exp, progenitors, schwann, pathol, axons,
Topics {✒️}
lysophosphatidylcholine-induced macrophage activation month download article/chapter tnf-alpha promotes proliferation interleukin-1beta promotes repair bone marrow cells experimentally-induced demyelinating lesions chronic-stage multiple sclerosis common neural progenitor acute demyelinating/remyelinating lesion macrophage-depletion induced impairment post-mitotic oligodendrocytes incapable glial growth factor-2 gliotoxin-induced cns demyelination oligodendrocyte progenitor cell ludwin sk privacy choices/manage cookies demyelinated spinal cord oligodendrocyte progenitor cells demyelinated rat axons oligodendrocyte precursor cells smith kj oligodendrocyte progenitor recruitment immune-mediated models bone morphogenetic proteins rat spinal cord sim fj device instant download early inflammatory responses rabbit spinal cord central nervous system experimental demyelinating diseases woodruff rh myelinated axons demonstrated repair demyelinated lesions chronically demyelinated lesions kotter mr glial cell transplants cre-mediated recombination minocycline-mediated inhibition trapidil-mediated inhibition enhancing oligodendrocyte survival diphtheria toxin fragment adult rat cns cell depletion due adult human brain experimental demyelinating lesions ethidium bromide injection keirstead hs transplanted glial cells glial cell environments
Schema {🗺️}
ScholarlyArticle:
headline:Remyelination in Experimental Models of Toxin-Induced Demyelination
pageEnd:212
pageStart:193
image:https://media.springernature.com/w153/springer-static/cover/book/978-3-540-73677-6.jpg
genre:
Biomedical and Life Sciences
Biomedical and Life Sciences (R0)
isPartOf:
name:Advances in multiple Sclerosis and Experimental Demyelinating Diseases
isbn:
978-3-540-73677-6
978-3-540-73676-9
type:Book
publisher:
name:Springer Berlin Heidelberg
logo:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
type:ImageObject
type:Organization
author:
name:W. F. Blakemore
affiliation:
name:University of Cambridge
address:
name:Department of Veterinary Medicine and Cambridge Centre for Brain Repair, University of Cambridge, Cambridge, UK
type:PostalAddress
type:Organization
type:Person
name:R. J. M. Franklin
affiliation:
name:University of Cambridge
address:
name:Department of Veterinary Medicine and Cambridge Centre for Brain Repair, University of Cambridge, Cambridge, UK
type:PostalAddress
type:Organization
type:Person
keywords:Experimental Autoimmune Encephalomyelitis, Schwann Cell, Myelin Sheath, Oligodendrocyte Progenitor Cell, Dorsal Funiculus
description:Remyelination is the regenerative process by which demyelinated axons are reinvested with new myelin sheaths. It is associated with functional recovery and maintenance of axonal health. It occurs as a spontaneous regenerative response following demyelination in a range of pathologies including traumatic injury as well as primary demyelinating disease such as multiple sclerosis (MS). Experimental models of demyelination based on the use of toxins, while not attempting to accurately mimic a disease with complex etiology and pathogenesis such as MS, have nevertheless proven extremely useful for studying the biology of remyelination. In this chapter, we review the main toxin models of demyelination, drawing attention to their differences and how they can be used to study different aspects of remyelination. We also describe the optimal use of these models, highlighting potential pitfalls in interpretation, and how remyelination can be unequivocally recognized. Finally, we discuss the role of toxin models alongside viral and immune-mediated models of demyelination.
datePublished:2008
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name:University of Cambridge
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name:Department of Veterinary Medicine and Cambridge Centre for Brain Repair, University of Cambridge, Cambridge, UK
type:PostalAddress
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name:Department of Veterinary Medicine and Cambridge Centre for Brain Repair, University of Cambridge, Cambridge, UK
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