DOI . ORG {
}
Title[redir]:
Studying Drug Resistance Using Genetically Engineered Mouse Models for Breast Cancer | SpringerLink
Description:
The generation of genetically engineered mouse models (GEMMs) that mimic breast cancer in humans provides new tools to investigate mechanisms of drug resistance in vivo. The advantages are manifold: inbred mice do not have the genomic heterogeneity seen in patients;...
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Keywords {🔍}
cancer, pubmed, google, scholar, article, cas, resistance, breast, drug, mouse, models, rottenberg, jonkers, tumors, rev, genetically, engineered, mammary, nat, privacy, cookies, content, information, publish, research, multidrug, protocol, therapy, search, pajic, molecular, mice, cells, access, borst, apoptosis, nature, proc, natl, acad, sci, usa, data, log, journal, sven, jos, biology, mechanisms, tumor,
Topics {✒️}
springer science+business media human brca1-mutated basal protocol multi-drug resistance marina pajic & jos jonkers brca1-deficient mammary tumors multi-drug resistance privacy choices/manage cookies studying drug resistance conditional mouse models humana press van der gulden gottesman mm reversing drug resistance drug resistance phenotype journal finder publish cancer drug resistance cancer drug development accelerated preclinical testing cancer sven rottenberg breast cancer protocol targeting multidrug resistance mimic breast cancer hereditary breast cancer mammalian abc transporters schmitt ca parp inhibitor azd2281 spontaneous immune responses conditional mouse model dutch cancer society netherlands cancer institute vivo pharmacology models conditions privacy policy author information authors editor information editors european economic area efficient tumour formation bmc cancer 6 anti-angiogenic therapy accepting optional cookies modeling therapy resistance chapter log main content log mouse models online protocol rottenberg jos jonkers drug resistance journal publish sven rottenberg ludwig ja
Questions {❓}
- De Visser KE (2008) Spontaneous immune responses to sporadic tumors: tumor-promoting, tumor-protective or both?
Schema {🗺️}
ScholarlyArticle:
headline:Studying Drug Resistance Using Genetically Engineered Mouse Models for Breast Cancer
pageEnd:45
pageStart:33
image:https://media.springernature.com/w153/springer-static/cover/book/978-1-60761-416-6.jpg
genre:
Springer Protocols
isPartOf:
name:Multi-Drug Resistance in Cancer
isbn:
978-1-60761-416-6
978-1-60761-415-9
type:Book
publisher:
name:Humana Press
logo:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
type:ImageObject
type:Organization
author:
name:Sven Rottenberg
affiliation:
name:The Netherlands Cancer Institute
address:
name:Division of Molecular Biology, The Netherlands Cancer Institute, Amsterdam, The Netherlands
type:PostalAddress
type:Organization
email:[email protected]
type:Person
name:Marina Pajic
affiliation:
name:The Netherlands Cancer Institute
address:
name:Division of Molecular Biology, The Netherlands Cancer Institute, Amsterdam, The Netherlands
type:PostalAddress
type:Organization
type:Person
name:Jos Jonkers
affiliation:
name:The Netherlands Cancer Institute
address:
name:Division of Molecular Biology, The Netherlands Cancer Institute, Amsterdam, The Netherlands
type:PostalAddress
type:Organization
email:[email protected]
type:Person
keywords:GEMM, Breast cancer, Orthotopic transplantation, BRCA1, BRCA2, E-cadherin, ATP-binding cassette (ABC) transporter, FACS sorting, Tumor-initiating cells
description:The generation of genetically engineered mouse models (GEMMs) that mimic breast cancer in humans provides new tools to investigate mechanisms of drug resistance in vivo. The advantages are manifold: inbred mice do not have the genomic heterogeneity seen in patients; mammary tumors are superficial and therefore easily accessible for measurement and sampling pre- and posttreatment; tumors can be transplanted orthotopically into syngeneic, immunocompetent animals; and tumor cells can be modified in vitro (e.g., gene overexpression, shRNA knockdown, insertional mutagenesis) prior to transplantation. Here, we provide an overview with experimental details of various approaches to study mechanisms of drug resistance in GEMMs for breast cancer.
datePublished:2010
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Book:
name:Multi-Drug Resistance in Cancer
isbn:
978-1-60761-416-6
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name:Humana Press
logo:
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type:ImageObject
name:The Netherlands Cancer Institute
address:
name:Division of Molecular Biology, The Netherlands Cancer Institute, Amsterdam, The Netherlands
type:PostalAddress
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name:Division of Molecular Biology, The Netherlands Cancer Institute, Amsterdam, The Netherlands
type:PostalAddress
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affiliation:
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address:
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type:PostalAddress
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name:Marina Pajic
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address:
name:Division of Molecular Biology, The Netherlands Cancer Institute, Amsterdam, The Netherlands
type:PostalAddress
type:Organization
name:Jos Jonkers
affiliation:
name:The Netherlands Cancer Institute
address:
name:Division of Molecular Biology, The Netherlands Cancer Institute, Amsterdam, The Netherlands
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email:[email protected]
PostalAddress:
name:Division of Molecular Biology, The Netherlands Cancer Institute, Amsterdam, The Netherlands
name:Division of Molecular Biology, The Netherlands Cancer Institute, Amsterdam, The Netherlands
name:Division of Molecular Biology, The Netherlands Cancer Institute, Amsterdam, The Netherlands
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