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  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Schema
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers
  13. CDN Services

We began analyzing https://link.springer.com/chapter/10.1007/82_2011_142, but it redirected us to https://link.springer.com/chapter/10.1007/82_2011_142. The analysis below is for the second page.

Title[redir]:
Roles of Lineage-Determining Transcription Factors in Establishing Open Chromatin: Lessons From High-Throughput Studies | SpringerLink
Description:
The interpretation of the regulatory information of the genome by sequence-specific transcription factors lies at the heart of the specification of cellular identity and function. While most cells in a complex metazoan organism express hundreds of such transcription...

Matching Content Categories {📚}

  • Education
  • Telecommunications
  • Science

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Doi.org Make Money? {💸}

We're unsure how the site profits.

Not all websites focus on profit; some are designed to educate, connect people, or share useful tools. People create websites for numerous reasons. And this could be one such example. Doi.org could be secretly minting cash, but we can't detect the process.

Keywords {🔍}

google, scholar, article, pubmed, cas, cell, transcription, factors, chromatin, biol, gene, cells, chapter, binding, expression, factor, mol, transcriptional, regulation, dev, stem, nature, development, glass, regulatory, genome, enhancers, information, open, heinz, access, human, liu, epub, brown, privacy, cookies, content, data, publish, epigenetic, regions, genes, genomic, control, dna, reprogramming, immunity, macrophages, drosophila,

Topics {✒️}

enhancer-driven lineage-specific transcription active cis-regulatory elements lineage-determining transcription factors month download article/chapter neuronal activity-regulated enhancers chip-seq experiments obtained /ebpalpha/beta convert fibroblasts blood stem/progenitor cells high-throughput studies chapter tissue-specific transcription genome-wide myod binding transcription factor access cell-specific determinants establishing open chromatin open chromatin enables gene regulatory networks chip reveals biniou chapter epigenetic regulation gene expression programs human cell types privacy choices/manage cookies transcription factor binding transcription factor activity predicting expression patterns antigen receptor diversity device instant download liver gene expression combinatorial transcriptional control conditions privacy policy innate immune response high-throughput studies transcription factor tal1 secondary transcription factors irf4 transcription factors hnf transcription factors predictive chromatin signatures histone modifications editor information editors early chromatin unfolding core transcriptional network epigenetic switch involved enhancer-based mechanisms cooperative dna binding disparate transcriptional activators pluripotent stem cells 3d chromatin organization european economic area animal body plans pasca di magliano miranda-carboni ga

Schema {🗺️}

ScholarlyArticle:
      headline:Roles of Lineage-Determining Transcription Factors in Establishing Open Chromatin: Lessons From High-Throughput Studies
      pageEnd:15
      pageStart:1
      image:https://media.springernature.com/w153/springer-static/cover/book/978-3-642-24103-1.jpg
      genre:
         Biomedical and Life Sciences
         Biomedical and Life Sciences (R0)
      isPartOf:
         name:Epigenetic Regulation of Lymphocyte Development
         isbn:
            978-3-642-24103-1
            978-3-642-24102-4
         type:Book
      publisher:
         name:Springer Berlin Heidelberg
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Sven Heinz
            affiliation:
                  name:University of California, San Diego
                  address:
                     name:Department of Cellular and Molecular Medicine, University of California, San Diego, La Jolla, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Christopher K. Glass
            affiliation:
                  name:University of California, San Diego
                  address:
                     name:Department of Cellular and Molecular Medicine, University of California, San Diego, La Jolla, USA
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
      keywords:Open Chromatin, Histone H3K4, Ternary Complex Formation, Histone Modification Pattern, Transcription Factor Motif
      description:The interpretation of the regulatory information of the genome by sequence-specific transcription factors lies at the heart of the specification of cellular identity and function. While most cells in a complex metazoan organism express hundreds of such transcription factors, the underlying mechanisms by which they ultimately achieve their functional locations within different cell types remain poorly understood. Here, we contrast various models of how cell type-specific binding patterns may arise using available evidence from ChIP-Seq experiments obtained in tractable developmental model systems, particularly the hematopoietic system. The data suggests a model whereby relatively small sets of lineage-determining transcription factors jointly compete with nucleosomes to establish their cell type-specific binding patterns. These binding sites gain histone marks indicative of active cis-regulatory elements and define a large fraction of the enhancer-like regions differentiated cell types. The formation of these regions of open chromatin enables the recruitment of secondary transcription factors that contribute additional transcription regulatory functionality required for the cell type-appropriate expression of genes with both general and specialized cellular functions.
      datePublished:2011
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
      context:https://schema.org
Book:
      name:Epigenetic Regulation of Lymphocyte Development
      isbn:
         978-3-642-24103-1
         978-3-642-24102-4
Organization:
      name:Springer Berlin Heidelberg
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:University of California, San Diego
      address:
         name:Department of Cellular and Molecular Medicine, University of California, San Diego, La Jolla, USA
         type:PostalAddress
      name:University of California, San Diego
      address:
         name:Department of Cellular and Molecular Medicine, University of California, San Diego, La Jolla, USA
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:Sven Heinz
      affiliation:
            name:University of California, San Diego
            address:
               name:Department of Cellular and Molecular Medicine, University of California, San Diego, La Jolla, USA
               type:PostalAddress
            type:Organization
      name:Christopher K. Glass
      affiliation:
            name:University of California, San Diego
            address:
               name:Department of Cellular and Molecular Medicine, University of California, San Diego, La Jolla, USA
               type:PostalAddress
            type:Organization
      email:[email protected]
PostalAddress:
      name:Department of Cellular and Molecular Medicine, University of California, San Diego, La Jolla, USA
      name:Department of Cellular and Molecular Medicine, University of California, San Diego, La Jolla, USA
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

External Links {🔗}(242)

Analytics and Tracking {📊}

  • Google Tag Manager

Libraries {📚}

  • Clipboard.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
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CDN Services {📦}

  • Pbgrd

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