NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC8100607/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC8100607/. The analysis below is for the second page.
Title[redir]:
Reporters of Cancer Stem Cells as a Tool for Drug Discovery - PMC
Description:
In view of the importance of cancer stem cells (CSCs) in chemoresistance, metastasis and recurrence, the biology of CSCs were explored in detail. Based on that, several modalities were proposed to target them. In spite of the several clinical ...
Title[redir]:
Reporters of Cancer Stem Cells as a Tool for Drug Discovery - PMC
Description:
In view of the importance of cancer stem cells (CSCs) in chemoresistance, metastasis and recurrence, the biology of CSCs were explored in detail. Based on that, several modalities were proposed to target them. In spite of the several clinical ...
Matching Content Categories {📚}
- Science
- Health & Fitness
- Education
Content Management System {📝}
What CMS is ncbi.nlm.nih.gov built with?
Custom-built
No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.
Traffic Estimate {📈}
What is the average monthly size of ncbi.nlm.nih.gov audience?
🌠 Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.
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How Does Ncbi.nlm.nih.gov Make Money? {💸}
We find it hard to spot revenue streams.
Not all websites are made for profit; some exist to inform or educate users. Or any other reason why people make websites. And this might be the case. Ncbi.nlm.nih.gov could have a money-making trick up its sleeve, but it's undetectable for now.
Keywords {🔍}
doi, cancer, cells, pubmed, google, scholar, cscs, stem, cell, pmc, drug, article, free, breast, abcg, aldha, csc, reporter, oct, metastasis, sox, expression, tumor, resistance, metastatic, molecules, targeting, signaling, cancers, human, reporters, markers, selfrenewal, pathways, clinical, screening, stemness, properties, leukemia, res, drugs, marker, oncol, resistant, target, nanog, population, reported, myeloid, aldh,
Topics {✒️}
cd133/cd44/oct4/nanog/sox2/abcb1/abcg2/abcc1 regulates emt/β-catenin/stt3 axis wnt/beta-catenin/myc/sox2 axis t315i-bcr-abl leukemia cells ccaat/enhancer-binding protein beta stt3-dependent pd-l1 accumulation mmtv-pymt mammary tumor model akt/nf-kappab/mdr1 pathway human colon-cancer-initiating cells shorter disease-free intervals lin−cd90−aldhhi cells mark major retinoic-acid-synthesizing enzyme small-cell lung carcinoma disulfiram/copper complex inhibits pmc beta search triple-negative breast cancer large b-cell lymphoma phase ii/iii trial stroma-induced drug tolerance notch3-expressing breast cancer enhanced pd-l1 output nanog inhibits β-catenin wnt/beta-catenin signaling cd44+cd24+aldh1+ subpopulation show ovarian cancer-initiating cells cancer stem-cell functions tgf-β signaling pathways cd133 regulates β-catenin high-throughput adaptable strategy hif-1α-dependent manner microenvironment-induced de-differentiation modified lentivirus-based reporter tumor-initiating cd49f cells lin−cd90+cd24+ cells cd44/cd105/cd106 high-throughput screening platforms oct4 promoter-mediated activity successful csc-targeting drug abcg2-meditated multidrug resistance lentiviral nanog-gfp reporter cd66+ cells drives nanog-egfp reporter system akt signal pathway oct4-egfp reporter system induces epithelial-mesenchymal transition cancer cells possessing leukaemia inhibitory factor cell stem cell brain cancer cells octamer-binding protein 4
Questions {❓}
- Bcl2 Inhibition by Venetoclax: Targeting the Achilles’ Heel of the Acute Myeloid Leukemia Stem Cell?
Social Networks {👍}(6)
External Links {🔗}(356)
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