NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC7067809/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC7067809/. The analysis below is for the second page.
Title[redir]:
Molecular principles of metastasis: a hallmark of cancer revisited - PMC
Description:
Metastasis is the hallmark of cancer that is responsible for the greatest number of cancer-related deaths. Yet, it remains poorly understood. The continuous evolution of cancer biology research and the emergence of new paradigms in the study of ...
Title[redir]:
Molecular principles of metastasis: a hallmark of cancer revisited - PMC
Description:
Metastasis is the hallmark of cancer that is responsible for the greatest number of cancer-related deaths. Yet, it remains poorly understood. The continuous evolution of cancer biology research and the emergence of new paradigms in the study of ...
Matching Content Categories {📚}
- Health & Fitness
- Science
- Education
Content Management System {📝}
What CMS is ncbi.nlm.nih.gov built with?
Custom-built
No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.
Traffic Estimate {📈}
What is the average monthly size of ncbi.nlm.nih.gov audience?
🌠 Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 6,398,976 visitors per month in the current month.
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How Does Ncbi.nlm.nih.gov Make Money? {💸}
We don’t know how the website earns money.
While many websites aim to make money, others are created to share knowledge or showcase creativity. People build websites for various reasons. This could be one of them. Ncbi.nlm.nih.gov might be cashing in, but we can't detect the method they're using.
Keywords {🔍}
doi, cancer, pubmed, google, scholar, cells, metastasis, pmc, article, free, cell, tumor, metastatic, breast, nature, nat, invasion, signaling, immune, role, exosomes, dormancy, ctcs, progression, microenvironment, emt, biol, metastases, growth, expression, ecm, rev, lung, bone, primary, matrix, survival, potential, addition, circulating, tumors, patients, factors, migration, target, promote, promotes, process, colonization, protein,
Topics {✒️}
regulating tweak/fn14/pi3k/akt pathway receptor-interacting serine/threonine-protein kinase epstein-barr virus-infected cells elicits t-cell-mediated attacks tissue-engineered tumor-microvessel platform cyclic gmp-amp synthase–stimulator regulating mir-142-5p/pik3ca axis platelet-derived growth factor-bb mcc-spain case-control study long-term follow-ups platelet-derived mhc class �weaning-induced liver involution interferon-gamma-mediated antitumor response il-6-stat3-saa signaling prevents targeting ezh2-stat3 signaling small leucine-rich proteoglycan mapk/beta-catenin axis astrocyte-derived exosomes mediate pi3k-akt signaling cascades herpesvirus increases pd-l1 pmc beta search live-cell fluorescence microscopy long-term tumor growth il-6/jak/stat3 pathway gain fat-lose metastasis transforming growth factor-β ras–mek–erk/mapk ebv-positive gastric cancer tumor-draining lymph nodes easing anchorage-independent survival facilitating anchorage-independent survival primary tumors pre-encode pre-metastatic niche formation tumor-derived extracellular vesicles semileptonic decay d0→a0 endothelial-cell-specific deletion wnt/β-catenin signaling weak cell–ecm adhesion hpv-positive cervical cancer cell-free dna extracellular matrix-derived peptides small leucine-rich proteoglycans autocrine wnt-pcp signaling secreted tumor-derived factors microenvironment-induced pten loss brings epithelial–mesenchymal transition directly targeting beta-catenin confer metastasis-initiating abilities extracellular signal‐regulated kinase immune cell-recognizable molecules
Questions {❓}
- Active versus passive mechanisms in metastasis: do cancer cells crawl into vessels, or are they pushed?
- Are circulating tumor cells target-specific?
- Can metastasis be driven by epigenetic factors?
- Can neurons initiate metastasis?
- Can surgical intervention contribute to metastatic dissemination?
- Diagnostics in circulation: where are we now?
- Do circulating tumor cells interact with immune cells?
- Does the circadian cycle play a role in tumorigenesis?
- Does the immune environment at the primary site play a role in metastasis?
- Does tumour dormancy offer a therapeutic target?
- Dormancy: why do cancer cells go to sleep?
- Epithelial-mesenchymal transition (EMT) and metastasis: yes, no, maybe?
- Epithelial–mesenchymal transition: what is new?
- From dormancy to activation: how does the dormant cell wake up?
- How do circulating tumor cells extravasate?
- How do tumor cells survive in circulation?
- How does autophagy contribute to cancer cell invasion?
- How does dormancy occur in metastatic clusters?
- How does the colonizing cell overcome stromal challenges?
- How does the microbiome contribute to cancer metastasis?
- How does the release of chemokines and cytokines help circulating tumor cells?
- Is tumor cell circulation contingent on the route of the bloodstream?
- Mechanisms of cancer cell dormancy—another hallmark of cancer?
- Small leucine-rich proteoglycans and matrix metalloproteinase-14: Key partners?
- Targeting circulating tumor cells: can it be done?
- What are the mechanisms that govern dormancy?
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