NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC6626132/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC6626132/. The analysis below is for the second page.
Title[redir]:
AMPA receptor GluA2 subunit defects are a cause of neurodevelopmental disorders - PMC
Description:
AMPA receptors (AMPARs) are tetrameric ligand-gated channels made up of combinations of GluA1-4 subunits encoded by GRIA1-4 genes. GluA2 has an especially important role because, following post-transcriptional editing at the Q607 site, it renders ...
Title[redir]:
AMPA receptor GluA2 subunit defects are a cause of neurodevelopmental disorders - PMC
Description:
AMPA receptors (AMPARs) are tetrameric ligand-gated channels made up of combinations of GluA1-4 subunits encoded by GRIA1-4 genes. GluA2 has an especially important role because, following post-transcriptional editing at the Q607 site, it renders ...
Matching Content Categories {📚}
- Education
- Science
- Books & Literature
Content Management System {📝}
What CMS is ncbi.nlm.nih.gov built with?
Custom-built
No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.
Traffic Estimate {📈}
What is the average monthly size of ncbi.nlm.nih.gov audience?
🌠 Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 6,732,313 visitors per month in the current month.
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How Does Ncbi.nlm.nih.gov Make Money? {💸}
We see no obvious way the site makes money.
Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Ncbi.nlm.nih.gov might be cashing in, but we can't detect the method they're using.
Keywords {🔍}
doi, glua, gria, supplementary, pubmed, variants, google, scholar, patient, mutations, pmc, article, free, individuals, recruited, study, denovo, patients, fig, cells, mutation, data, disorders, analysis, panel, variant, current, identified, receptor, genes, including, ascertained, sequencing, residue, dee, channels, neurodevelopmental, subunits, affected, research, nat, ampa, mutant, coexpressed, rectification, molecular, channel, genet, site, results,
Topics {✒️}
include n-methyl-d-aspartate receptors anti-hebbian long-term potentiation thiamine deficiency-induced alteration ca/cam-dependent kinase ii ampa receptor-stargazin complexes competing interests calcium-permeable ampa receptors amber99sb-ildn62 force field ampa-subtype glutamate receptor similar nmdar-independent form hand-operated piezo manipulator ampa-subtype glutamate receptors web-based tools genematcher31 eliminates ka-evoked currents20 poly-d-lysine pmc beta search ampa-type glutamate receptors exhibiting hand-wringing suggestive bio-rad image lab single-nucleotide substitution leads de-novo deleterious mutations gria2 de-novo variants greater paired-end reads alternative flip/flop splicing26 dominant de-novo mutations ka-evoked current amplitude identify de-novo mutations independent t-tests epilepsy-aphasia spectrum disorders license information pmcid peer review information lrfn2/salm1-deficient mice linear current-voltage relationship22 rare de-novo mutations agonist-evoked current mediated gria2-related ndd occurred uk/tools/msa/clustalw2/ wild-type crystal structure custom-developed analysis tool57 molecular inversion probes adar2-dependent rna editing author contributions protein-coding genetic variation holm-bonferroni p data availability statements data availability statement human glua2 wild-type ionotropic glutamate receptors genomic glutamine-encoding codon ampa receptor tetramerization
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