Here's how DOI.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
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DOI . ORG {}

Detected CMS Systems:

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Wordpress Themes And Plugins
  7. Keywords
  8. Topics
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://journals.plos.org/plosgenetics/article?id=10.1371/journal.pgen.1006129, but it redirected us to https://journals.plos.org/plosgenetics/article?id=10.1371/journal.pgen.1006129. The analysis below is for the second page.

Title[redir]:
No title found...
Description:
Author Summary Rett syndrome (RTT) is a debilitating neurodevelopmental disorder with no cure or effective treatment. To fully understand the disease mechanism and develop therapies, it is necessary to study all aspects of the molecular function of methyl-CpG binding protein 2 (MeCP2), mutations in which have been identified as the genetic cause of RTT. Over the years, MeCP2 has been shown to maintain DNA methylation, regulate transcription and chromatin structure, control microRNA processing, and modulate RNA splicing. Among these known functions, the role of MeCP2 in modulating RNA splicing is less well understood. We took several unbiased approaches to investigate the how MeCP2 may regulate splicing, what splicing changes are caused by the loss of MeCP2, and what functional consequences are caused by altered splicing. We discovered that MeCP2 interacts with splicing factors to regulated the splicing of glutamate receptor genes, which mediate the vast majority of excitatory synaptic transmission in the brain; and linked the altered splicing of glutamate receptor genes to specific synaptic changes in a RTT mouse model. Our findings not only advance the understanding of RTT disease mechanism, but also reveal a potential drug target for future development of therapies.

Matching Content Categories {📚}

  • Science
  • Education
  • Virtual Reality

Content Management System {📝}

What CMS is doi.org built with?


Doi.org is powered by WORDPRESS. But there are also traces of other content systems on the page (hubspot).

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌍 Impressive Traffic: 500k - 1M visitors per month


Based on our best estimate, this website will receive around 600,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Doi.org Make Money? {💸}

We can't see how the site brings in money.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Doi.org might be plotting its profit, but the way they're doing it isn't detectable yet.

Wordpress Themes and Plugins {🎨}

What WordPress theme does this site use?

It is strange but we were not able to detect any theme on the page.

What WordPress plugins does this website use?

It is strange but we were not able to detect any plugins on the page.

Keywords {🔍}

mecp, splicing, gria, article, view, pmid, pubmedncbi, google, scholar, fig, mice, flipflop, analysis, data, cortex, mouse, genes, epub, central, pubmed, pmcid, brain, rna, alternative, gene, ledgf, identified, exon, rtt, proteins, study, flip, interaction, neurons, events, protein, cells, chipseq, exons, pmc, altered, flop, dna, sem, genome, cell, ratio, synaptic, chromatin, western,

Topics {✒️}

human health cell biology aav-esf-flop-gly-mcherry significantly altered swi/snf chromatin-remodeling complex plos genet 12 plos genet cookies plos myc-tagged full-length ledgf/p52 n-terminal pwwp-cr1 domain plos esf-flop-gly expressing neurons inherited mental retardation esf-flop-gly [flop suppressor] methyl-cpg-binding protein methyl-cpg binding protein-2 esf-flop-sr [flop enhancer] esf-flip-gly [flip suppressor] esf-flip-sr [flip enhancer] human molecular genetics mek/erk-regulated cell proliferation methyl-cpg-binding domain antibody-proteing-bead complex expresses flag-tagged mecp2 esf-flop-gly significantly increased glutamate-evoked ampar-gated current als-linked proteins fus/tls swi/snf complex regulates pre-mrna splicing esf-flop-gly didn overexpressing esf-flop-gly conducted high-throughput sequencing global epigenomic reconfiguration aav-esf-flop-gly nature genetics esf-flop-gly lentivirus original author histone deacetylase complex global splicing alterations processing long pre-mrnas li-cor odyssey imager ledgf/p75-binding abilities mecp2-flag knockin mice specific protein-protein interactions reversing flip/flop splicing spinal muscular atrophy flip/flop splicing defect dna methylation data alternative pre-mrna splicing maintain dna methylation maintaining dna methylation[10]

External Links {🔗}(332)

Analytics and Tracking {📊}

  • Facebook Pixel
  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager
  • Google Universal Analytics
  • HubSpot

Libraries {📚}

  • Backbone.js
  • Foundation
  • jQuery
  • Leaflet
  • Lightbox
  • Modernizr
  • Moment.js
  • Prism.js
  • Underscore.js
  • Video.js
  • Vue.js
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {📦}

  • Cloudflare
  • Crossref

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