Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC5747297/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC5747297/. The analysis below is for the second page.

Title[redir]:
Monocyte-Macrophages and T Cells in Atherosclerosis - PMC
Description:
Atherosclerosis is an arterial disease process characterized by the focal subendothelial accumulation of apolipoprotein B-lipoproteins, immune and vascular wall cells, and extracellular matrix. The lipoproteins acquire features of damage-associated ...

Matching Content Categories {📚}

  • Science
  • Health & Fitness
  • Education

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We can't see how the site brings in money.

Not all websites focus on profit; some are designed to educate, connect people, or share useful tools. People create websites for numerous reasons. And this could be one such example. Ncbi.nlm.nih.gov could be getting rich in stealth mode, or the way it's monetizing isn't detectable.

Keywords {🔍}

doi, cells, google, scholar, pubmed, atherosclerosis, pmc, cell, article, free, macrophages, inflammatory, mice, inflammation, plaque, treg, macrophage, atherosclerotic, cholesterol, disease, immune, monocytes, resolution, lesional, lesions, studies, response, activation, progression, increased, study, lesion, expression, plaques, proliferation, wang, efferocytosis, metabolism, lipid, function, regulatory, promotes, functions, differentiation, van, res, nature, vascular, effector, receptor,

Topics {✒️}

cd4+ccr5+ifnγ+foxp3+t-bet+ cells ccr5+t-bet+foxp3+ effector cd4 western diet-fed lnk-/-ldlr-/ lineage-specific t-bet deletion western diet-fed ldlr-/ ccr5+ foxp3+t-bet+ phenotype anti-il-1β blocking antibody atp-binding cassette transporters treg-cell-restricted abcg1 deficiency nuclear factor-kappab-mediated inflammation western-type diet rich il-1β-induced il-6 production hif-1α-deficient apcs express activates macrophage nf-κb produce abundant ifn-γ transcription factor t-bet cd11c-cre-mediated deletion il-3-gm-csf receptor signaling pmc beta search protein-p38 mapk pathway adam17-mediated mertk cleavage transcription factors t-bet src/pi3k/rac1 signaling mtor-mediated stat5 signaling c-reactive protein lipopolysaccharide-induced endotoxic shock inhibits tnf-alpha production including inflammasome-derived il-1β hmg-coa reductase inhibitor low-grade chronic inflammation regulates monocyte/macrophage proliferation ifn-γ-producing interleukin-1β-converting enzyme thrombosis-induced organ infarction block hif-1α translation oxidize glucose-derived pyruvate amp-activated protein kinase glycolysis-induced pd-l1 plastic ifnγ+ th1/tregs unlike anti-inflammatory drugs glut1-mediated glucose uptake heart immediately post-mi bone marrow-derived monocytes ly-6chigh monocytes depend pre-existing lesional monocytes aforementioned plaque-stabilizing functions checkpoint inhibitor pd-l1 physiologic post-inflammatory response meyer zu hg pro-atherogenic interleukin 1β

Questions {❓}

  • Expansion of necrotic core and shedding of Mertk receptor in human carotid plaques: a role for oxidized polyunsaturated fatty acids?
  • Macrophage immunometabolism: Where are we (going)?
  • Monocyte count: a risk factor for coronary heart disease?
  • Novel lipid mediators and resolution mechanisms in acute inflammation: to resolve or not?
  • Once a Treg, always a Treg?

Social Networks {👍}(6)

External Links {🔗}(312)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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