Here's how NCBI.NLM.NIH.GOV makes money* and how much!

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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC5663297/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC5663297/. The analysis below is for the second page.

Title[redir]:
Impact of Oxidative Stress on the Heart and Vasculature Part 2 of a 3-Part Series - PMC
Description:
Vascular disease and heart failure impart an enormous burden in terms of global morbidity and mortality. Although there are many different causes of cardiac and vascular disease, most share an important pathological mechanism: oxidative stress. In ...

Matching Content Categories {📚}

  • Health & Fitness
  • Fitness & Wellness
  • Education

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 6,399,052 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We can't figure out the monetization strategy.

While profit motivates many websites, others exist to inspire, entertain, or provide valuable resources. Websites have a variety of goals. And this might be one of them. Ncbi.nlm.nih.gov might be earning cash quietly, but we haven't detected the monetization method.

Keywords {🔍}

doi, pubmed, google, scholar, ros, vascular, oxidative, stress, endothelial, mitochondrial, free, pmc, nadph, article, oxidase, dysfunction, heart, disease, mice, atherosclerosis, production, role, antioxidant, nox, nitric, oxide, enos, biol, oxygen, cardiovascular, activation, mitochondria, superoxide, hypertension, synthase, failure, cardiac, redox, increased, patients, human, circulation, res, function, activity, protein, cell, cells, expression, factors,

Topics {✒️}

low-dose alpha-tocopherol improves c-jun n-terminal kinases inducible nitric-oxide synthase peroxisome proliferator-activated receptor-α oxidized low-density lipoprotein nicotinamide adenine dinucleotide manganese-dependent superoxide dismutase angiotensin ii-induced production african-american heart failure cu/zn-superoxide dismutase angiotensin ii-induced hypertension zinc [cu-zn]-sod gq-coupled angiotensin ii low-density lipoprotein renin-angiotensin-aldosterone systems calcium-calmodulin–dependent activation angiotensin ii-mediated hypertension human αb-crystallin mutation streptozotocin-induced hyperglycemic mice sod effect dose-dependent plant-derived polyphenol resveratrol renin-angiotensin-aldosterone system blood-brain barrier damage include receptor-induced activation involves redox-sensitive kinases angiotensin-converting-enzyme inhibitor advanced glycation end-products human αb-crystallin gene decreases leukocyte-endothelial interactions enos substrate l-arginine partner site rhein-main redox-sensitive transcription factors pmc beta search doxorubicin-induced heart damage redox-dependent signaling processes endothelium-derived vasoactive substances blood pressure-lowering effects hydrogen peroxide ubiquinone-cytochrome b-c1 pressure overload-induced hf age-dependent vascular dysfunction age-dependent enos uncoupling angiotensin-converting enzyme inhibitors at1-type angiotensin receptor mitochondrial na+/ca2+ exchanger detrimental ros-induced remodeling myocardial ischemia/reperfusion injury reactive oxygen species-studies apolipoprotein e-deficient mice anti-oxidant systems distributed

Questions {❓}

  • , Jr Are ACE inhibitors a “magic bullet” against oxidative stress?
  • Heme oxygenase-1: a novel drug target for atherosclerotic diseases?
  • How does ascorbic acid prevent endothelial dysfunction?
  • Is oxidative stress a therapeutic target in cardiovascular disease?
  • Is the oxidative modification hypothesis relevant to human atherosclerosis?
  • Reactive oxygen species, vascular oxidative stress, and redox signaling in hypertension: what is the clinical significance?
  • The antioxidant paradox: less paradoxical now?

External Links {🔗}(348)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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