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Keywords {🔍}

aip, pubmed, google, scholar, doi, cell, pmc, article, free, caspase, cells, proliferation, drosophila, death, dronc, caspases, apoptosis, tumor, apoptotic, cancer, regeneration, jnk, activity, figure, activation, signaling, compensatory, mechanisms, biol, caspasedependent, tissue, production, required, wound, response, model, development, healing, functions, effector, apoptosisinduced, dev, bergmann, studies, ros, role, repopulation, nonapoptotic, caspasedriven, immune,

Topics {✒️}

cysteine-dependent aspartate-directed proteases pmc beta search reaper-grim-hid-mediated suppression elif kamber kaya reduces caspase-driven apoptosis nab-brk signal bifurcates calcium-insensitive phospholipase a2 diap1-dependent dronc ubiquitination card-carrying caspase dronc desired anti-tumor functions cyclooxygenase-dependent tumor growth stress-responsive promoter element ras-dependent survival signaling undesired pro-tumor functions post-irradiation angiogenesis e3-ligase drosophila inhibitor de-ubiquitylating enzyme duba caspase-dependent tumor repopulation ros-dependent proliferation pathway ca2+-independent phospholipase a2 caspase-driven compensatory proliferation limited pro-apoptotic stimulus similar caspase/pge2 mechanisms caspase-inhibited apoptotic cells multiple upstream pro-apoptotic initiator caspase-dependent model apoptosis-induced proliferation pathways jnk-dependent apoptosis triggered apoptosis-induced compensatory proliferation dronc-mediated aip depends cell death-induced regeneration anti-vegf/vegfr therapy reactive oxygen species jnk/dronc feedback amplification bmp/tgfβ homolog decapentaplegic requires additional input effector caspase-dependent aip caspase-3-dependent aip acting effector caspase-driven aip pro-apoptotic genes reaper caspase-3-activated signaling pathways scaffolding protein dark/apaf-1 bcl-2 protein family multiple hematopoietic lineages tumour cell proliferation dvorak hf hepatocyte cell death caspase-dependent aip mechanisms modulation factor directing references haynie jl

Questions {❓}

• Are cell types other than epithelial cells subject to AiP?
• Are there key regulatory points in these caspase-dependent pathways that can be targeted, to distinguish between the various pleiotropic effects, enhancing the desired anti-tumor functions while suppressing the undesired pro-tumor functions?
• Does AiP contribute to other disease processes?
• How general is the involvement of AiP in tumor development?
• How is this distinction regulated?
• Multiple mechanisms of AiP: redundancy or context-specific?
• Regulating the targets: a role for adaptors and scaffolds directing AiP versus apoptotic death?
• What are the direct targets of caspases in AiP?
• Why does activation of caspases in apoptosis sometimes induce AiP and other times not?

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• https://www.linkedin.com/company/ncbinlm
• https://twitter.com/nlm_nih
• https://www.facebook.com/nationallibraryofmedicine
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