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We began analyzing https://link.springer.com/article/10.1007/BF00848779, but it redirected us to https://link.springer.com/article/10.1007/BF00848779. The analysis below is for the second page.

Title[redir]:
The effects of X-rays on the proliferation dynamics of cells in the imaginal wing disc ofDrosophila melanogaster | Discover Developmental Biology
Description:
We report on the size distribution of clones marked by mitotic recombination induced by several different doses of X-rays applied to 72 h oldDrosophila larvae. The results indicate that the radiation significantly reduces the number of cells which undergo normal proliferation in the imaginal wing disc. We estimate that 1000 r reduces by 40โ€“60% the number of cells capable of making a normal contribution to the development of the adult wing. Part of this reduction is due to severe curtailment in the proliferative ability of cells which nevertheless remain capable of adult differentiation; this effect is possibly due to radiation-induced aneuploidy. Cytological evidence suggests that immediate cell death also occurs as a result of radiation doses as low as 100 r. The surviving cells are stimulated to undergo additional proliferation in response to the X-ray damage so that the result is the differentiation of a normal wing.

Matching Content Categories {๐Ÿ“š}

  • Education
  • Science
  • Telecommunications

Content Management System {๐Ÿ“}

What CMS is doi.org built with?

Custom-built

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Traffic Estimate {๐Ÿ“ˆ}

What is the average monthly size of doi.org audience?

๐Ÿ™๏ธ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 80,479,999 visitors per month in the current month.

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Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Doi.org could have a money-making trick up its sleeve, but it's undetectable for now.

Keywords {๐Ÿ”}

google, scholar, melanogaster, imaginal, ofdrosophila, cell, cells, wing, discs, wilhelm, developmental, biology, article, disc, radiation, effects, bryant, development, indrosophila, garciabellido, biol, pattern, privacy, cookies, rouxs, archives, xrays, proliferation, haynie, roux, archiv, content, publish, search, download, drosophila, growth, develop, formation, schneiderman, york, springer, nature, analysis, data, information, journal, research, john, larvae,

Topics {โœ’๏ธ}

mitotic recombination induced x-chromosome ofdrosophila melanogaster imaginal wing disc related subjects imaginal discs ofdrosophila haltere imaginal discs cells indrosophila melanogaster irradiation ofdrosophila melanogaster privacy choices/manage cookies cell death cytoskeletal regulation cell lineage relationships undergo additional proliferation main content log wilhelm roux' archiv115 wilhelm roux' archiv134 wilhelm roux' archiv172 wilhelm roux' archiv166 ofdrosophila melanogaster imaginal discs developmental analysis european economic area radiation-induced aneuploidy cytological evidence suggests past caspase activity show reduced loss biological radiotracer methods ciba foundation symposium29 x-rays indrosophila undergo normal proliferation developmental biology aims x-ray damage conditions privacy policy drosophila somatic pairing derived development ofdrosophila x-rays applied accepting optional cookies usage analysis june 1977 volumeย 183 radiation significantly reduces cell division journal finder publish ๏ฟฝcell-lethalsโ€ article haynie usa john search search normal wing cell biology cell lineage

Schema {๐Ÿ—บ๏ธ}

WebPage:
      mainEntity:
         headline:The effects of X-rays on the proliferation dynamics of cells in the imaginal wing disc ofDrosophila melanogaster
         description:We report on the size distribution of clones marked by mitotic recombination induced by several different doses of X-rays applied to 72 h oldDrosophila larvae. The results indicate that the radiation significantly reduces the number of cells which undergo normal proliferation in the imaginal wing disc. We estimate that 1000 r reduces by 40โ€“60% the number of cells capable of making a normal contribution to the development of the adult wing. Part of this reduction is due to severe curtailment in the proliferative ability of cells which nevertheless remain capable of adult differentiation; this effect is possibly due to radiation-induced aneuploidy. Cytological evidence suggests that immediate cell death also occurs as a result of radiation doses as low as 100 r. The surviving cells are stimulated to undergo additional proliferation in response to the X-ray damage so that the result is the differentiation of a normal wing.
         datePublished:
         dateModified:
         pageStart:85
         pageEnd:100
         sameAs:https://doi.org/10.1007/BF00848779
         keywords:
            Pattern regulation
            Cell death
             Drosophila
            Imaginal discs
            Clonal analysis
            Mitotic recombination
            Developmental Biology
            Evolutionary Biology
            Zoology
            Animal Genetics and Genomics
            Cell Biology
         image:
         isPartOf:
            name:Wilhelm Roux's archives of developmental biology
            issn:
               1432-041X
               0340-0794
            volumeNumber:183
            type:
               Periodical
               PublicationVolume
         publisher:
            name:Springer-Verlag
            logo:
               url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
               type:ImageObject
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         author:
               name:John L. Haynie
               affiliation:
                     name:University of California
                     address:
                        name:Department of Developmental and Cell Biology and Center for Pathobiology, University of California, Irvine, USA
                        type:PostalAddress
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                     address:
                        name:Department of Developmental and Cell Biology and Center for Pathobiology, University of California, Irvine, USA
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ScholarlyArticle:
      headline:The effects of X-rays on the proliferation dynamics of cells in the imaginal wing disc ofDrosophila melanogaster
      description:We report on the size distribution of clones marked by mitotic recombination induced by several different doses of X-rays applied to 72 h oldDrosophila larvae. The results indicate that the radiation significantly reduces the number of cells which undergo normal proliferation in the imaginal wing disc. We estimate that 1000 r reduces by 40โ€“60% the number of cells capable of making a normal contribution to the development of the adult wing. Part of this reduction is due to severe curtailment in the proliferative ability of cells which nevertheless remain capable of adult differentiation; this effect is possibly due to radiation-induced aneuploidy. Cytological evidence suggests that immediate cell death also occurs as a result of radiation doses as low as 100 r. The surviving cells are stimulated to undergo additional proliferation in response to the X-ray damage so that the result is the differentiation of a normal wing.
      datePublished:
      dateModified:
      pageStart:85
      pageEnd:100
      sameAs:https://doi.org/10.1007/BF00848779
      keywords:
         Pattern regulation
         Cell death
          Drosophila
         Imaginal discs
         Clonal analysis
         Mitotic recombination
         Developmental Biology
         Evolutionary Biology
         Zoology
         Animal Genetics and Genomics
         Cell Biology
      image:
      isPartOf:
         name:Wilhelm Roux's archives of developmental biology
         issn:
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            0340-0794
         volumeNumber:183
         type:
            Periodical
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            type:ImageObject
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      author:
            name:John L. Haynie
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                  name:University of California
                  address:
                     name:Department of Developmental and Cell Biology and Center for Pathobiology, University of California, Irvine, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Peter J. Bryant
            affiliation:
                  name:University of California
                  address:
                     name:Department of Developmental and Cell Biology and Center for Pathobiology, University of California, Irvine, USA
                     type:PostalAddress
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["Periodical","PublicationVolume"]:
      name:Wilhelm Roux's archives of developmental biology
      issn:
         1432-041X
         0340-0794
      volumeNumber:183
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      name:Springer-Verlag
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
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      name:University of California
      address:
         name:Department of Developmental and Cell Biology and Center for Pathobiology, University of California, Irvine, USA
         type:PostalAddress
      name:University of California
      address:
         name:Department of Developmental and Cell Biology and Center for Pathobiology, University of California, Irvine, USA
         type:PostalAddress
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      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:John L. Haynie
      affiliation:
            name:University of California
            address:
               name:Department of Developmental and Cell Biology and Center for Pathobiology, University of California, Irvine, USA
               type:PostalAddress
            type:Organization
      name:Peter J. Bryant
      affiliation:
            name:University of California
            address:
               name:Department of Developmental and Cell Biology and Center for Pathobiology, University of California, Irvine, USA
               type:PostalAddress
            type:Organization
PostalAddress:
      name:Department of Developmental and Cell Biology and Center for Pathobiology, University of California, Irvine, USA
      name:Department of Developmental and Cell Biology and Center for Pathobiology, University of California, Irvine, USA

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