
NCBI . NLM . NIH . GOV {
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Title[redir]:
Oncostatin M promotes cancer cell plasticity through cooperative STAT3-SMAD3 signaling - PMC
Description:
Increasing evidence supports the idea that cancer cell plasticity promotes metastasis and tumor recurrence, resulting in patient mortality. While it is clear that the tumor microenvironment (TME) contributes to cancer cell plasticity, the specific ...
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Keywords {🔍}
osm, cells, smad, scholar, pubmed, google, doi, cancer, stat, cell, pmc, article, csc, free, figure, cdcd, expression, breast, signaling, exposure, tgfβ, properties, tumor, emt, plasticity, growth, mesenchymalcsc, cytokines, analysis, epithelialnoncsc, tme, nuclear, gene, protein, usa, activation, transition, exposed, weeks, control, factor, phosphorylation, cytokine, assessed, epithelialmesenchymal, acquisition, osminduced, population, genes, snail,
Topics {✒️}
atm-chk2-p53-independent ras-induced senescence tgf-beta-induced epithelial-mesenchymal transition [pubmed] [google scholar] opts tgf-β/smad signaling tgf-beta-induced interleukin-6 participates tgf-β/smad3 cooperatively engage tgf-β/smad cross-talk pmc beta search tgf-beta signaling engages single-gene qrt-pcr analysis promote stat3/smad3-mediated transcription transforming growth factor-β tgf-β-mediated il-6 production counteract polycomb-mediated repression granulocyte-colony stimulating factor osm-mediated mesenchymal/csc properties tgf-beta superfamily signaling tgf-β-induced snail induction bone marrow x-linked functional tgf-β signaling tight cell–cell interactions van boxel-dezaire ah epithelial-mesenchymal transition markers cd24−/cd44+ mesenchymal/csc population fluorouracil-based adjuvant chemotherapy tgf-beta-induced epithelial tgf-β-responsive genes tgf-β/smad signaling tgf-β receptor ii canonical jak/stat3 signaling integrated data-mining platform nuclear-soluble protein fractions il-1-stimulated human fibroblasts tgf-β receptor activity protein tyrosine kinases/phosphatases activates canonical jak/stat3 mek–erk-dependent manner tgf-β receptor type osmr/jak-mediated phosphorylation tgf-β signaling contributes increase tgfβr-mediated phosphorylation tgf-β/smad3 signaling tgf-β-induced genes expressing dominant-negative stat3 epithelial-mesenchymal transition phenotype triple-negative breast cancer promote epithelial–mesenchymal plasticity cooperative stat3-smad3 signaling stat3/smad3 signaling interactions smad-target gene serpine1
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