NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC4898967/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC4898967/. The analysis below is for the second page.
Title[redir]:
TREM2 haplodeficiency in mice and humans impairs the microglia barrier function leading to decreased amyloid compaction and severe axonal dystrophy - PMC
Description:
Haplodeficiency of the microglia gene TREM2 increases risk for late-onset Alzheimerโs disease (AD) but the mechanisms remain uncertain. To investigate this, we used high-resolution confocal and super-resolution (STORM) microscopy in AD-like mice and ...
Title[redir]:
TREM2 haplodeficiency in mice and humans impairs the microglia barrier function leading to decreased amyloid compaction and severe axonal dystrophy - PMC
Description:
Haplodeficiency of the microglia gene TREM2 increases risk for late-onset Alzheimerโs disease (AD) but the mechanisms remain uncertain. To investigate this, we used high-resolution confocal and super-resolution (STORM) microscopy in AD-like mice and ...
Matching Content Categories {๐}
- Education
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Content Management System {๐}
What CMS is ncbi.nlm.nih.gov built with?
Custom-built
No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.
Traffic Estimate {๐}
What is the average monthly size of ncbi.nlm.nih.gov audience?
๐ Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 6,398,976 visitors per month in the current month.
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How Does Ncbi.nlm.nih.gov Make Money? {๐ธ}
We're unsure how the site profits.
The purpose of some websites isn't monetary gain; they're meant to inform, educate, or foster collaboration. Everyone has unique reasons for building websites. This could be an example. Ncbi.nlm.nih.gov might be plotting its profit, but the way they're doing it isn't detectable yet.
Keywords {๐}
microglia, trem, plaques, doi, mice, plaque, amyloid, figure, processes, pubmed, google, scholar, barrier, pmc, fibrils, filamentous, compact, article, dap, free, human, dystrophy, brain, individual, axonal, disease, deficiency, function, surface, degree, images, diffuse, alzheimers, increased, quantification, confocal, observed, thioflavin, fibril, mutations, xfad, total, humans, neurites, compaction, condello, core, imaging, risk, storm,
Topics {โ๏ธ}
n-terminus anti-aฮฒ antibody genome-wide gene-expression analysis light-induced dark states {beta}-stimulated microglial activation pmc beta search experimental procedures animals post-mortem temporal cortices multicolor time-stamp reveals abeta42-driven cerebral amyloidosis 642nm solid-state laser large late-onset family super-resolution storm imaging neuritic tau hyper-phosphorylation performed super-resolution nanoscopy positron emission tomography late-onset human ad intraneuronal beta-amyloid aggregates cd68-immunolabeled microglial phagosomes atomic-force microscopy data middle frontal gyrus postmortem human brain human brain tissue hyper-phosphorylated dystrophic neurites lamp1-immunolabeled axonal dystrophy fibrils extending radially common nasu-hakola disease 50mm sodium citrate postmortem human brains high-resolution confocal poor antibody penetration strong anti-colocalization human brain tissues dap12-deficient mice fail higher protofibrillar aฮฒ42 olmos-alonso iba1-labeled surrounding microglia cross-section fluorescence intensity human ad tissue mouse somatosensory cortex increased orthogonal branches branched aฮฒ nanostructures human tissue staining altered microglial response examined microglia-plaque interactions reactive oxygen species impaired antigen priming supplementary materials included rapid plaque growth single aฮฒ fibrils atomic-force microscopy
Social Networks {๐}(6)
External Links {๐}(113)
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