Here's how NCBI.NLM.NIH.GOV makes money* and how much!

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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3690135/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC3690135/. The analysis below is for the second page.

Title[redir]:
Regulation of the inflammatory response in cardiac repair - PMC
Description:
Myocardial necrosis triggers an inflammatory reaction that clears the wound from dead cells and matrix debris, while activating reparative pathways necessary for scar formation. A growing body of evidence suggests that accentuation, prolongation or ...

Matching Content Categories {📚}

  • Health & Fitness
  • Education
  • Science

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 6,399,052 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

The income method remains a mystery to us.

While profit motivates many websites, others exist to inspire, entertain, or provide valuable resources. Websites have a variety of goals. And this might be one of them. Ncbi.nlm.nih.gov might be plotting its profit, but the way they're doing it isn't detectable yet.

Keywords {🔍}

doi, inflammatory, pubmed, google, scholar, inflammation, myocardial, infarction, response, cells, role, signaling, heart, infarcted, pmc, resolution, postinfarction, tgfβ, cell, remodeling, cardiac, article, free, regulation, receptor, activation, growth, injury, infarct, suppression, expression, mediators, myocardium, effects, cytokine, chemokine, proinflammatory, signals, endothelial, matrix, pathways, neutrophils, acute, socs, negative, patients, mice, protein, immunol, responses,

Topics {✒️}

advanced glycation end-products macrophage-colony stimulating factor intercellular adhesion molecule m-csf reactive oxygen species tumor necrosis factor nuclear factor autophagy-related molecule atg16l1 targeting c-reactive protein monocyte chemoattractant protein-1/ccl2 transforming growth factor-beta inhibiting nf-κb activation c-reactive protein junctional adhesion molecules vcam jam transforming growth factor interleukin-1 receptor antagonist monocyte chemoattractant protein-1 species-specific differences nf-κb growth differentiation factor icam-1/p-selectin-knockout mice higher peak tnf-α advanced coronary care biosynthetic intermediate 17s-hydroxy-dha leukocyte/endothelial cell adhesion stimulating synthesis receptor 3-induced nf-kappa tgf-β-mediated myofibroblast differentiation il-1 receptor antagonist growth factor production ros production serum amyloid peptide junctional locations leading duffy antigen receptor duffy antigen/receptor α-smooth muscle actin mapk phosphatase developmental endothelial locus high-mobility group box-1 tgf-beta-induced smad signaling dipeptidyl peptidase interleukin-1 receptor type endogenous il-1ra tumor myeloid cell marker ros jak-independent suppressive actions activate jak/stat

Questions {❓}

  • Considering the extensive evidence supporting their effectiveness in experimental animal models, what is the basis for failure of targeted anti-inflammatory strategies in reducing ischemic cardiac injury?
  • However, to support the therapeutic importance of this concept, a key question needs to be answered: to what extent is defective negative regulation of the inflammatory response responsible for adverse remodeling and worse outcome in patients with acute myocardial infarction?
  • Novel lipid mediators and resolution mechanisms in acute inflammation: to resolve or not?
  • The clinical implications: is adverse remodeling in patients with myocardial infarction due to defective suppression and impaired resolution of inflammation?

Social Networks {👍}(6)

External Links {🔗}(267)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • AOS
  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

4.25s.