NCBI . NLM . NIH . GOV {
}
We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3672955/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC3672955/. The analysis below is for the second page.
Title[redir]:
TH17 cell plasticity in Peyer’s patches is responsible for induction of T cell-dependent IgA responses - PMC
Description:
Intestinal Peyer’s patches are essential lymphoid organs for the generation of T cell-dependent immunoglobulin (Ig) A production for gut homeostasis. Using IL-17 fate reporter mice we show here that endogenous TH17 cells in lymphoid organs of naïve ...
Title[redir]:
TH17 cell plasticity in Peyer’s patches is responsible for induction of T cell-dependent IgA responses - PMC
Description:
Intestinal Peyer’s patches are essential lymphoid organs for the generation of T cell-dependent immunoglobulin (Ig) A production for gut homeostasis. Using IL-17 fate reporter mice we show here that endogenous TH17 cells in lymphoid organs of naïve ...
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- Science
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What CMS is ncbi.nlm.nih.gov built with?
Custom-built
No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.
Traffic Estimate {📈}
What is the average monthly size of ncbi.nlm.nih.gov audience?
🌠 Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 6,694,437 visitors per month in the current month.
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How Does Ncbi.nlm.nih.gov Make Money? {💸}
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While many websites aim to make money, others are created to share knowledge or showcase creativity. People build websites for various reasons. This could be one of them. Ncbi.nlm.nih.gov could be secretly minting cash, but we can't detect the process.
Keywords {🔍}
cells, mice, doi, eyfp, iga, tfh, expression, pubmed, google, scholar, tcra, cell, intestinal, pmc, ilacrerreyfp, free, treg, article, cholera, germinal, cxcr, data, foxp, toxin, helper, center, transferred, plasticity, responses, phenotype, response, lymphoid, gut, transfer, flow, hosts, rorc, follicular, proportion, chimeras, induction, panel, immunol, reporter, intestine, development, small, profile, cytometry, nature,
Topics {✒️}
tgf-beta-induced foxp3 inhibits germ-free surrogate mothers pmc beta search cholera toxin-specific iga antigen-specific iga response antigen-specific iga responses antigen-specific intestinal iga cholera toxin-immunized mice activation-induced cytidine deaminase identifying foxp3-expressing suppressor specific pathogen free t-independent iga-producing cell-independent iga synthesis iga-producing germinal center regulatory cell-iga response wild-type reporter mice cell-dependent iga-secreting cell-dependent iga responses cxcr5+ pd-1high cells wild-type il17acrer26reyfp mice immune wild-type mice cell-dependent iga production pathogen free conditions intact wild-type hosts supplementary materials included ectopic lymphoid follicles th17 cell-derived tfh bcl-6 requires icos38 cell-deficient tcra−/− mice cell-independent iga induction polymeric ig receptor wild-type b6 mice tcra-deficient mice transferred license information pmcid 25μg cholera toxin cholera toxin mice germinal center response cxcr5+ pd-1high class-switch recombination induce germinal center high rfp-foxp3 expression real-time pcr cell iga response list biological laboratories basal iga levels transcription factor bcl614-16 transcription factor rorα45 germinal center switch fate reporter mouse cholera toxin resulted
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- https://doi.org/10.1073/pnas.0501701102's revenue stream
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