Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3203524/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC3203524/. The analysis below is for the second page.

Title[redir]:
Leukocyte Complexity Predicts Breast Cancer Survival and Functionally Regulates Response to Chemotherapy - PMC
Description:
Immune-regulated pathways influence multiple aspects of cancer development. In this article we demonstrate that both macrophage abundance and T-cell abundance in breast cancer represent prognostic indicators for recurrence-free and overall survival. ...

Matching Content Categories {📚}

  • Science
  • Education
  • Health & Fitness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We can't see how the site brings in money.

Not all websites are made for profit; some exist to inform or educate users. Or any other reason why people make websites. And this might be the case. Ncbi.nlm.nih.gov could have a money-making trick up its sleeve, but it's undetectable for now.

Keywords {🔍}

cancer, doi, breast, mice, tumor, fig, cells, expression, mammary, tumors, google, scholar, pubmed, ptx, mmtvpymt, patients, supplementary, immune, treated, plx, article, tams, pmc, treatment, infiltration, tissue, cell, analysis, response, chemotherapy, tam, free, macrophages, survival, csf, data, mrna, macrophage, recruitment, significant, therapy, cohort, primary, lymphocytes, metastasis, signature, cytotoxic, density, antitumor, human,

Topics {✒️}

cd45+cd11blow/−ly6c−cd22−ly6g−cd11chighmhciihigh dcs tumor-infiltrating d45+ly6g−ly6clowcd11b+f4/80+ macrophages cd45+cd11b+ly6c−ly6g−f4/80+ tam recruitment tumor-infiltrating cd45+ly6g−ly6clowcd11b+f4/80+ tams cd45+cd11blow/−cd19−ly6g−ly6clowcd11chighmhciihigh dcs cd45+ly6g−ly6c−cd11b+f4/80+ tams cd45+cd11b+ly6c−ly6g−f4/80+ tams colony-stimulating factor-1-related proteins cd45+ly6g−ly6clowcd11blow/− cd11chighmhciihi dcs cd45+cd11b+ly6clowly6g−f4/80+ tams colony-stimulating factor-1 receptor oncogenic b-raf kinase cd45+cd11b+ly6g−ly6clowf4/80+ cd8+ t-cell–dependent manner mmtv-pmecs–derived mecs treated cd45+cd11b+ly6g−ly6chigh cd8+ t-cell–dependent response macrophage-deficient op/op mouse tumor-infiltrating cd45+cd3+cd8+ late-stage mmtv-pymt mice lin−/lowhla-dr−cd33+cd11b+ cd68low/cd4low/cd8high immune profiles part cd8+ t-cell–dependent cd8+ t-cell–dependent mechanisms cd68low/cd4low/cd8high immune signature pbs-perfused mmtv-pymt mice cd14+hla-dr−/low imcs cd11b+gr1+ly6g+ cells cd45+cd11b+ly6ghigh imcs cd45+cd11b+ly6ghi imcs brooks/cole advanced books orthotopic tumor–bearing mice orthotopic pymt-derived tumors cd14+hla-dr−/low cells cd8+ t-cell–dependent csf-1 receptor-mediated regulation immunosuppressive cd11b+ly6g+ cells myeloid-derived suppressor cells colony stimulating factor-1 colony-stimulating factor-1 colony-stimulating factor 1 cd45+cd11b+cd14+ macrophages rfs comparing cd68high/cd4high/cd8low cd68/cd4/cd8 immune signature cd8+ t-cell infiltration malignant pymt-derived mecs mmtv-pymt–derived mecs cd8 t-cell ratios gr1+ccr2+cx3cr1low imcs mmtv-pymt mice progress

Questions {❓}

  • M-CSF (monocyte colony stimulating factor) and M-CSF receptor expression by breast tumour cells: M-CSF mediated recruitment of tumour infiltrating monocytes?

External Links {🔗}(133)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

3.93s.