Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC2471394/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC2471394/. The analysis below is for the second page.

Title[redir]:
Bypassing cellular EGF receptor dependence through epithelial-to-mesenchymal-like transitions - PMC
Description:
Over 90% of all cancers are carcinomas, malignancies derived from cells of epithelial origin. As carcinomas progress, these tumors may lose epithelial morphology and acquire mesenchymal characteristics which contribute to metastatic potential. An ...

Matching Content Categories {๐Ÿ“š}

  • Science
  • Education
  • Health & Fitness

Content Management System {๐Ÿ“}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {๐Ÿ“ˆ}

What is the average monthly size of ncbi.nlm.nih.gov audience?

๐ŸŒ  Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Ncbi.nlm.nih.gov Make Money? {๐Ÿ’ธ}

We don't see any clear sign of profit-making.

Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Ncbi.nlm.nih.gov has a secret sauce for making money, but we can't detect it yet.

Keywords {๐Ÿ”}

doi, pubmed, google, scholar, cancer, cell, cells, growth, egfr, tumor, ecadherin, factor, emt, receptor, signaling, epithelial, expression, metastasis, shown, transition, igfr, erlotinib, pmc, survival, breast, mesenchymal, cellular, article, pathways, carcinoma, inhibition, res, activation, snail, transitions, epidermal, lines, free, biol, human, apoptosis, kinase, inhibitors, insulinlike, invasion, emtlike, progression, phenotype, transcriptional, sensitivity,

Topics {โœ’๏ธ}

ras-raf-mek-erk pathways igf-1r-irs1-akt axis platelet-derived growth factor igf-1r/pi3k signaling axis epidermal growth factor/hydrocortisone igf-1r/egfr cross-talks e-cadherin-repressing transcription factors pi3 kinase-akt-mtor pi 3-kinase/akt signaling pi3k-akt survival pathway pmc beta search igf-1r tyrosine kinase fibroblast-derived epithelial morphogen small-cell lung cancer ras-mapk proliferative pathway egf receptor/akt pathway regulating pro-apoptotic pathways zinc-finger transcription factors hormone-refractory prostate cancer bypassing egfr-dependent activation tight cellโ€“cell junctions downregulate endogenous e-cadherin affect e-cadherin repressors dominant negative mutant epidermal growth factor tgf-ฮฒ induced apoptosis blivet-van eggelpoel mj tgf-beta confers resistance igf-1r targeted therapeutics e-cadherin converts cells e-cadherin mutant cells e-cadherin gene expression epithelial-mesenchymal transition induced suppress e-cadherin expression high-grade prostate cancer growth factor receptor growth factor-1 receptor small molecule inhibitors survived tgf-ฮฒ treatment repressing e-cadherin expression trastuzumab-refractory breast cancer partial epithelial-mesenchymal transition small cell lung alternate growth factor increased igf-1r signaling igf-1r targeted therapies osi pharmaceuticals/genetech/roche publisher site pdf pi3 kinase pathway transitions igf-1r signaling

Questions {โ“}

  • Epidermal growth factor receptor dependence in human tumors: more than just expression?
  • Snail, Zeb and bHLH factors in tumour progression: an alliance against the epithelial phenotype?

External Links {๐Ÿ”—}(203)

Analytics and Tracking {๐Ÿ“Š}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {๐Ÿ“š}

  • jQuery
  • jQuery module (jquery-3.6.0)

Emails and Hosting {โœ‰๏ธ}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {๐Ÿ“ฆ}

  • Ncbi

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