
NCBI . NLM . NIH . GOV {
}
Detected CMS Systems:
- MyBB (1 occurrences)
Title[redir]:
Role of the nucleus in apoptosis: signaling and execution - PMC
Description:
Since their establishment in the early 1970s, the nuclear changes upon apoptosis induction, such as the condensation of chromatin, disassembly of nuclear scaffold proteins and degradation of DNA, were, and still are, considered as the essential ...
Matching Content Categories {π}
- Science
- Telecommunications
- Education
Content Management System {π}
What CMS is ncbi.nlm.nih.gov built with?
Ncbi.nlm.nih.gov is built with MYBB.
Traffic Estimate {π}
What is the average monthly size of ncbi.nlm.nih.gov audience?
π Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Ncbi.nlm.nih.gov Make Money? {πΈ}
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The purpose of some websites isn't monetary gain; they're meant to inform, educate, or foster collaboration. Everyone has unique reasons for building websites. This could be an example. Ncbi.nlm.nih.gov might be earning cash quietly, but we haven't detected the monetization method.
Keywords {π}
doi, pubmed, google, scholar, cell, dna, apoptosis, pmc, apoptotic, article, free, nuclear, pml, activation, death, caspase, proteins, damage, response, activity, cells, protein, fig, mdm, biol, stress, chromatin, function, degradation, cellular, atm, regulation, nbs, cancer, phosphorylation, daxx, nucleus, shown, signaling, rps, fragmentation, mechanisms, ribosomal, complex, caspases, rpl, role, induction, cycle, cleavage,
Topics {βοΈ}
c-jun amino-terminal kinase c-jun n-terminal kinase swi/snf protein atrx cd95/fas-dependent apoptosis signaling c-myc-promoted tumor growth promote cd95/fas-induced apoptosis pmc beta search bravo-san pedro jm cd95/fas-induced apoptosis due glucocorticoid-induced thymocyte apoptosis microhomology-mediated end joining c-abl phosphorylates hdmx cysteinyl aspartate-specific proteases n-terminal transcriptional domain cd95/fas death receptor acinus-provoked protein kinase large-scale dna fragmentation axin/tip60/hipk2/p53 complexes double-strand-specific dnase dna double-strand breaks sarco-er ca2+-atpase aberrant p53-induced apoptosis promyelocytic leukemia protein primes p53-mediated apoptosis cd95/fas-induced apoptosis stress dose-dependent manner double-strand break repair nur77-dependent apoptotic pathway p53-mediated apoptotic pathway pml-interacting protein daxx promyelocytic leukaemia protein prosurvival nuclear factor-ΞΊb p53-null mefs revealed cancer-derived p53 mutations single-strand break repair rich sequence-binding protein-1 atr/atr-interacting protein p53-deficient cells affected mitogen-activated protein kinases mitochondrial apoptosis-inducing factor atr-mediated checkpoint signaling caspase-9-null mice suggest suppress c-myc activity death-inducing signaling complex nuclear factor-kappab nur77-targeting compounds revealed extensive p53-mediated apoptosis promiscuously interacting protein p38mapk/mk2 kinase complex simultaneous caspase-3-mediated activation
Questions {β}
- Caspase-2: what do we know today?
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