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Keywords {🔍}

cabl, hdm, phosphorylation, mdm, cells, google, scholar, expression, wildtype, cell, activity, kinase, figure, response, protein, dna, lane, transfected, mutant, plasmids, levels, lanes, vitro, vivo, tyrosine, sionov, damage, vogt, degradation, antibody, crossref, nuclear, interaction, smp, kinasedefective, antibodies, haupt, substitution, fibroblasts, regulation, atm, antihdm, site, effect, results, ability, analysis, plasmid, role, western,

Topics {✒️}

anti‐phospho‐394y polyclonal antibodies wild‐type pcmv‐neo‐bam‐hdm2 share previous article anti‐phospho‐y394 polyclonal antibodies full‐length wild‐type hdm2 double‐strand dna breaks anti‐phosphoy394 polyclonal antibodies cooh‐terminal repeated domain israel science foundation anti‐phosphotyrosine monoclonal antibody author author contributions envision peroxidase anti‐mouse anti‐phospho‐ser15 antibody pi3‐kinase/akt pathway crossref dai-chi liu pcmv‐neo‐bam‐p53 mdm2 ring‐finger domain request permissions keywords u1a‐positive nuclear bodies pcmv‐neo‐bam‐hdm2 anti‐α‐tubulin dm1a methods cell culture download pdf hrp‐conjugated protein cisplatin‐induced dna damage crossref yi-fan wang nucleo‐cytoplasmic shuttling depends il lautenberg center p53/mdm2 2ko mice anti‐phospho‐y394 antibodies dna‐dependent protein kinase ubiquitin–proteasome‐mediated degradation molecular cell biology p53‐deficient embryo fibroblasts authors submit p53/mdm2 double knockout anti‐rabbit igg multi‐protein binding domain express endogenous wild‐type er stress-induced modulation embo journal vol dna damage‐induced apoptosis dna damage‐induced modification include wild‐type hdm2 c-abl activation downstream p53‐specific cdna probe cells expressing wild‐type ring‐finger domain e3‐ubiquitin ligase pcmv‐neo‐bam

Questions {❓}

• How can this apparent contradiction be resolved?
• In this study we ask whether this neutralization involves a direct interplay between c‐Abl and Mdm2, and what is the contribution of the c‐Abl kinase activity?
• Meek DW (1999) Mechanisms of switching on p53: a role for covalent modification?

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