PMC . NCBI . NLM . NIH . GOV {
}
We are analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3978912/.
Title:
Progesterone receptor assembly of a transcriptional complex along with activator protein 1, signal transducer and activator of transcription 3 and ErbB-2 governs breast cancer growth and predicts response to endocrine therapy - PMC
Description:
The role of the progesterone receptor (PR) in breast cancer remains a major clinical challenge. Although PR induces mammary tumor growth, its presence in breast tumors is a marker of good prognosis. We investigated coordinated PR rapid and ...
Website Age:
27 years and 8 months (reg. 1997-10-02).
Title:
Progesterone receptor assembly of a transcriptional complex along with activator protein 1, signal transducer and activator of transcription 3 and ErbB-2 governs breast cancer growth and predicts response to endocrine therapy - PMC
Description:
The role of the progesterone receptor (PR) in breast cancer remains a major clinical challenge. Although PR induces mammary tumor growth, its presence in breast tumors is a marker of good prognosis. We investigated coordinated PR rapid and ...
Website Age:
27 years and 8 months (reg. 1997-10-02).
Matching Content Categories {๐}
- Science
- Education
- Health & Fitness
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Custom-built
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๐ Phenomenal Traffic: 5M - 10M visitors per month
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How Does Pmc.ncbi.nlm.nih.gov Make Money? {๐ธ}
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Keywords {๐}
cells, doi, figure, cancer, cyclin, mpa, breast, cjun, tam, pubmed, google, scholar, promoter, nuclear, erbb, growth, expression, transcriptional, cfos, levels, receptor, effects, activation, tumor, colocalization, phosphorylation, cell, found, tumors, progesterone, pmc, protein, stat, article, results, pcjun, file, chd, binding, response, findings, free, complex, erฮฑ, human, additional, activity, progestin, analysis, mapks,
Topics {โ๏ธ}
c-src/p42/p44mapks pathway occurs gov/ij/docs/guide] bailey tl preventing c-src/p42/p44mapks activation lacking p-c-jun/pr colocalization ap-1/stat3/pr/erbb-2 complex assembled c-jun n-terminal kinase progestin-activated p42/p44 mapks src/p42/p44mapks cascade leading c-src/p42/p44mapks cascade tpa-modulated trans-acting factor tissue-selective agonist/antagonist activities pr-activated p42/p44 mapks c-jun/c-fos/estrogen receptor ap-1/stat3/pr/erbb-2 transcriptional complex activated c-src/p42/p44mapks study p-c-jun/pr colocalization growth-promoting ap-1/pr complex ligand-induced transcription factor including c-jun n-terminal antagonist-occupied steroid receptors enhance p-c-jun levels c4hd-tam-67-herbb-2ฮดnls tumors showed mitogen-activated protein kinases abstract introduction real-time quantitative pcr her2-positive breast cancersโrole src/p21ras/erk pathway carboxyl-terminal transactivation domain c-src/erk pathway human wild-type hpr activate p42/p44 mapks mpa-induced c-jun phosphorylation platelet-derived growth factor p-c-jun/pr colocalization control c4hd-p-flag cells abbreviations a-fos pmc beta search p42/p44 mapks activation vivo progestin-induced growth ap-1/stat3/pr/erbb-2 enhanceosome nuclear c-jun/pr colocalization phorbol ester-inducible genes c4hd-a-fos/herbb-2ฮดnls ap-1/stat3/erbb-2/pr complex analyzed p-c-jun expression pr/-c-fos nuclear interactions p42/p44 mapks phosphorylation c-fos/pr nuclear colocalization references daniel ar dana-farber cancer institute
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