PMC . NCBI . NLM . NIH . GOV {
}
We are analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3553257/.
Title:
Microglia of the Aged Brain: Primed to be Activated and Resistant to Regulation - PMC
Description:
Innate immunity within the central nervous system (CNS) is primarily provided by resident microglia. Microglia are pivotal in immune surveillance and also facilitate the coordinated responses between the immune system and the brain. For example, ...
Website Age:
27 years and 9 months (reg. 1997-10-02).
Title:
Microglia of the Aged Brain: Primed to be Activated and Resistant to Regulation - PMC
Description:
Innate immunity within the central nervous system (CNS) is primarily provided by resident microglia. Microglia are pivotal in immune surveillance and also facilitate the coordinated responses between the immune system and the brain. For example, ...
Website Age:
27 years and 9 months (reg. 1997-10-02).
Matching Content Categories {📚}
- Education
- Science
- Health & Fitness
Content Management System {📝}
What CMS is pmc.ncbi.nlm.nih.gov built with?
Custom-built
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Traffic Estimate {📈}
What is the average monthly size of pmc.ncbi.nlm.nih.gov audience?
🌠 Phenomenal Traffic: 5M - 10M visitors per month
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How Does Pmc.ncbi.nlm.nih.gov Make Money? {💸}
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Keywords {🔍}
doi, microglia, pubmed, google, scholar, aged, brain, mice, aging, microglial, pmc, article, free, inflammatory, increased, activation, expression, immune, lps, ilβ, adult, age, cns, response, rats, fractalkine, inflammation, peripheral, challenge, injection, receptor, exaggerated, neuroinflammation, cells, memory, cognitive, prolonged, compared, impairments, ltp, behavior, protein, antiinflammatory, effects, neurobiol, cytokines, deficits, hippocampal, lynch, impaired,
Topics {✒️}
dysregulated neuronal-microglial cross-talk nih grants r01-ag-033028 brain-derived neurotrophic factor bone-marrow derived cells high il-1β concentrations pmc beta search reduces beta-amyloid deposition anti-inflammatory agent resveratrol inhibit long-term potentiation transforming growth factor-β1 intramedullary prostaglandin-dependent mechanism toxin-induced demyelination lesion stress-related neuroendocrine circuitry potent anti-inflammatory cytokine growth hormone-dependent manner increased pro-inflammatory cytokines pro-inflammatory il-1beta ncam-derived mimetic peptide blood-born factors responsible cd200-deficient mice show intracellular il-1β production free radical damage pivotal pro-inflammatory genes anti-inflammatory agent rosiglitazone regulate il-1β production blood-brain barrier alterations lowering hippocampal il-1β anti-inflammatory il-10 cytokines reduces il-1β mrna bone marrow-derived including mhc ii stress-activated protein kinases enhanced il-1beta production amplified il-1β production pro-inflammatory cytokines tnfα decreased tnf-α response cytokine-induced sickness behavior intrahippocampal il-1beta administration neuropathol appl neurobiol recent article reports pre-clinical prion disease blocks il-1β signaling microglial cell-mediated neuroinflammation increased il-1β mrna transforming growth factor neuro–glia interaction effects long term potentiation long-term potentiation infection-induced memory deficits anti-inflammatory factor
Questions {❓}
- Immune activation in brain aging and neurodegeneration: too much or too little?
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