We are analyzing https://link.springer.com/article/10.1007/s00109-025-02528-x.

Title:
Targeting ferroptosis for the treatment of female reproductive system disorders | Journal of Molecular Medicine
Description:
Ferroptosis, a regulated form of cell death driven by iron-dependent lipid peroxidation, has emerged as a critical factor in female reproductive health and has been implicated in disorders such as polycystic ovary syndrome, premature ovarian insufficiency, endometriosis, and ovarian cancer. This review explores the intricate molecular mechanisms underlying ferroptosis, emphasizing its reliance on iron metabolism and oxidative stress, which disrupt key processes in reproductive tissues, including granulosa cell function, folliculogenesis, and embryo implantation. Increasing evidence linking ferroptosis to these conditions offers new therapeutic opportunities, with iron chelators, lipid peroxidation inhibitors, and antioxidants showing the potential to alleviate reproductive dysfunction by modulating ferroptotic pathways. In ovarian cancer, ferroptosis inducers combined with conventional cancer therapies, such as chemotherapy, provide promising strategies to overcome drug resistance. This review synthesizes current knowledge on ferroptosis and highlights its importance as a therapeutic target in reproductive health, emphasizing the need for further research to refine and expand treatment options, evaluate their applicability in clinical settings, and explore their role in fertility preservation. By advancing our understanding of ferroptosis regulation, these therapeutic approaches could lead to novel treatments for reproductive disorders and cancers, offering new hope for improving outcomes in women’s health and cancer therapy.
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28 years and 1 months (reg. 1997-05-29).

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Keywords {🔍}

ferroptosis, pubmed, google, scholar, cas, zhang, cancer, chen, cell, central, wang, ovarian, liu, iron, front, biol, zhao, yang, cells, tang, res, sci, article, reproductive, mol, kang, ovary, zhou, med, zhejiang, lipid, polycystic, syndrome, mao, death, metabolism, therapeutic, gpx, feng, medical, disorders, mechanisms, resistance, disease, chem, ferritinophagy, yan, province, medicine, oxidative,

Topics {✒️}

gpx4-gss/gsr-ggt axis activity doxorubicin-induced oxidative damage mir-761-hepcidin/gpx4 pathway contribute rna-binding protein elavl1/hur sepsis-induced cardiac injury hypoxia-inducible factor antisense nf-kb signaling pathway single-cell rna-sequencing analysis shi-liang chen & yi-bo masked nitrile-oxide electrophiles cerebral ischemia-induced ferroptosis month download article/chapter photo‐enhanced synergistic induction prevent hydroperoxide-induced ferroptosis small-cell lung carcinoma ferroptosis-resistant cell state female reproductive health hiv-infected individuals revealed chemotherapy-induced ovarian failure reproductive disorders ferroportin-mediated iron accumulation kras-mutant lung adenocarcinoma combined vegf/pd-1 blockade cancer treatment-induced cardiotoxicity oxidized low-density lipoproteins regulating tet activity long noncoding rnas ferroptosis signaling pathways cystine transporter slc7a11/xct subsequently increases il-1β cell-specific metabolic reprogramming nuclear receptor coactivator-4 iron-sulfur cluster proteins brca-proficient ovarian cancer iron-dependent lipid peroxidation cell death induced lipid-peroxidation burst device alleviate reproductive dysfunction maternal high-fat � long-circulating assembly glutathione-independent ferroptosis suppressor cisplatin-resistant ovarian cancer aging rat ovaries ovarian cancer cells—”utilization” targeting pten polycystic ovary syndrome ferroptosis-related genes oligodendrocyte ferroptosis induced ferroptosis alleviates atherosclerosis hydrogen peroxide triggers

Questions {❓}

Feng H, Stockwell BR (2018) Unsolved mysteries: how does lipid peroxidation cause ferroptosis?

Schema {🗺️}

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