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cells, cancer, cell, twist, breast, par, taz, expression, article, control, emt, hippo, pathway, invasion, google, scholar, cas, snail, vector, data, tumor, fig, experiments, migration, nature, metastasis, signaling, size, analysis, twistmediated, ecadherin, performed, transition, overexpression, activation, ctgf, tdtwist, epithelialmesenchymal, promotes, increased, expressing, presented, wound, assay, sirna, luciferase, wang, growth, epithelial, gene,

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nature portfolio recent research shows privacy policy cancer research nature hela cdna library editing advertising social media 0/ reprints confer csc-related traits speckle-type poz protein pkcepsilon-dependent mechanism snail-mediated e-cadherin repression genome chip-chip analysis twist-expressing t47d cells gsk-3beta-mediated phosphorylation full size image twist-t47d cells compared undergone twist-mediated emt providing ctgf-luc plasmid inflammation-induced cell migration twist-overexpressing t47d cells g-protein-coupled receptors par1/gα13 binding conformation t47d-twist cell types normal breast epithelia twist-mediated par1 induction mesenchymal marker n-cadherin epithelial marker e-cadherin subsequently activates yap/taz confers tumor cells epithelial-mesenchymal transition epithelial–mesenchymal transition national cancer institute-frederick cellular de-differentiation program matrigel-coated boyden chambers high-gain sensors breast cancer progression stem cell traits human breast cancer permissions real-time pcr luciferase reporter assay mammary epithelial cells twist promotes induction twist-expression affects proliferation cancer stem cell twist-expressing t47d stable cell line

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