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nature portfolio journals permissions reprints nature portfolio privacy policy advertising health research akt/gsk3-beta signaling pathway social media national library research grants hyperglycemia-induced podocyte injury nature+ nature 454 nature 467 nature set7/9-induced histone methylation inositol-requiring enzyme 1α inositol-requiring enzyme-1α dahl salt-sensitive rat complement-mediated glomerular injury n-linked glycosylation sites lead-induced oxidative stress protein-tyrosine phosphatase 1b gov/ct2/show/nct02343094 er stress-induced autophagy albumin overload-induced nephropathy ameliorate protein-folding diseases x-box binding protein-1 personal data ischemia-reperfusion injury author information authors permissions attenuates kidney injury erse-ii element hypoxic conditions stimulate endoplasmic reticulum stress endoplasmic-reticulum stress prevent neurodegeneration nf-κb-mediated inflammation α-actinin-4-mediated fsgs springerlink instant access data protection acute ischemic injury renal tubular injury glomerular basement membrane inhibits cell proliferation acute kidney injury transcription factor xbp-1 impairs cytoskeletal dynamics chop/gadd153 repression

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• Stress in the kidney is the road to pERdition: is endoplasmic reticulum stress a pathogenic mediator of diabetic nephropathy?
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