Here's how NATURE.COM makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

NATURE . COM {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Nature.com Make Money
  6. How Much Does Nature.com Make
  7. Keywords
  8. Topics
  9. Schema
  10. Social Networks
  11. External Links
  12. Analytics And Tracking
  13. Libraries
  14. Hosting Providers
  15. CDN Services

We are analyzing https://www.nature.com/articles/ncomms1909.

Title:
Trim71 cooperates with microRNAs to repress Cdkn1a expression and promote embryonic stem cell proliferation | Nature Communications
Description:
Pluripotent embryonic stem cells have a shortened cell cycle that enables their rapid proliferation. The embryonic stem cell-specific miR-290 and miR-302 microRNA families promote proliferation whereas let-7 microRNAs inhibit self-renewal, and promote cell differentiation. Lin28 suppresses let-7 expression in embryonic stem cells. Here to gain further insight into mechanisms controlling embryonic stem cell self-renewal, we explore the molecular and cellular role of the let-7 target Trim71 (mLin41). We show that Trim71 associates with Argonaute2 and microRNAs, and represses expression of Cdkn1a, a cyclin-dependent kinase inhibitor that negatively regulates the G1–S transition. We identify protein domains required for Trim71 association with Argonaute2, localization to P-bodies, and for repression of reporter messenger RNAs. Trim71 knockdown prolongs the G1 phase of the cell cycle and slows embryonic stem cell proliferation, a phenotype that was rescued by depletion of Cdkn1a. Thus, we demonstrate that Trim71 is a factor that facilitates the G1–S transition to promote rapid embryonic stem cell self-renewal. Embryonic stem cells have a shortened cell cycle that allows for rapid proliferation, but the exact mechanisms are unclear. Here, a microRNA target, Trim71, is shown to inhibit the expression of a cyclin-dependent kinase inhibitor, thus enabling the G1–S phase cell cycle transition in embryonic stem cells.
Website Age:
30 years and 10 months (reg. 1994-08-11).

Matching Content Categories {πŸ“š}

  • Video & Online Content
  • Education
  • DIY & Home Improvement

Content Management System {πŸ“}

What CMS is nature.com built with?

Custom-built

No common CMS systems were detected on Nature.com, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of nature.com audience?

πŸŒ† Monumental Traffic: 20M - 50M visitors per month


Based on our best estimate, this website will receive around 42,556,742 visitors per month in the current month.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Nature.com Make Money? {πŸ’Έ}


Display Ads {🎯}


The website utilizes display ads within its content to generate revenue. Check the next section for further revenue estimates.

Ads are managed by yourbow.com. Particular relationships are as follows:

Direct Advertisers (10)
google.com, pmc.com, doceree.com, yourbow.com, audienciad.com, onlinemediasolutions.com, advibe.media, aps.amazon.com, getmediamx.com, onomagic.com

Reseller Advertisers (38)
conversantmedia.com, rubiconproject.com, pubmatic.com, appnexus.com, openx.com, smartadserver.com, lijit.com, sharethrough.com, video.unrulymedia.com, google.com, yahoo.com, triplelift.com, onetag.com, sonobi.com, contextweb.com, 33across.com, indexexchange.com, media.net, themediagrid.com, adform.com, richaudience.com, sovrn.com, improvedigital.com, freewheel.tv, smaato.com, yieldmo.com, amxrtb.com, adyoulike.com, adpone.com, criteo.com, smilewanted.com, 152media.info, e-planning.net, smartyads.com, loopme.com, opera.com, mediafuse.com, betweendigital.com

How Much Does Nature.com Make? {πŸ’°}


Display Ads {🎯}

$536,300 per month
Estimations show Nature.com's display ad online revenue falls between $357,519 and $983,176 per month.

Keywords {πŸ”}

trim, cell, escs, pubmed, cells, article, cdkna, ago, fig, expression, lin, scholar, google, mirnas, cas, control, stem, proliferation, reporter, levels, protein, mouse, cycle, proteins, nature, rna, repression, esc, mirna, central, embryonic, domain, found, sirna, nhl, assays, differentiation, target, interaction, luciferase, data, knockdown, gene, performed, reverse, selfrenewal, activity, transfected, localization, utr,

Topics {βœ’οΈ}

pew research scholar nature portfolio permissions reprints privacy policy advertising social media generate pflag-tnrc6b c-term32 ultraviolet-crosslinked dox-inducible escs nature 463 nature 454 nature 462 nature pflag-ms2-tnrc6b c-term full access multiple small-rna-mediated pathways high-affinity ms2-binding sites quantitative real-time pcr extended population-doubling time33 cyclin e-cdk2 complex34 tumor suppressor mei-p26 mirna-mediated gene regulation23 dox-inducible flag-trim71 escs cyclin e-cdk2 complex c57bl/6j genomic dna pflag-tnrc6b c-term ms2-tnrc6b c-terminus Ξ±-flag m2-peroxidase open biosystems author correspondence impairs p-body localization post-transcriptional gene repression unique cell-cycle structure es cell-specific mirna post-transcriptional gene silencing n-terminal flag-tag neuroprogenitor-based genetic subtype development mirna-induced silencing complex full size image edta-free bc-100 buffer edta-free bc buffer induce mirna-mediated repression embryonic stem cell pflag-trim71 full-length nonsense-mediated mrna decay mei-p26 regulates micrornas trim71-overexpressing cell populations amino-terminal ring finger post-transcriptionally repress expression n-terminal truncations interact

Schema {πŸ—ΊοΈ}

WebPage:
      mainEntity:
         headline:Trim71 cooperates with microRNAs to repress Cdkn1a expression and promote embryonic stem cell proliferation
         description:Pluripotent embryonic stem cells have a shortened cell cycle that enables their rapid proliferation. The embryonic stem cell-specific miR-290 and miR-302 microRNA families promote proliferation whereas let-7 microRNAs inhibit self-renewal, and promote cell differentiation. Lin28 suppresses let-7 expression in embryonic stem cells. Here to gain further insight into mechanisms controlling embryonic stem cell self-renewal, we explore the molecular and cellular role of the let-7 target Trim71 (mLin41). We show that Trim71 associates with Argonaute2 and microRNAs, and represses expression of Cdkn1a, a cyclin-dependent kinase inhibitor that negatively regulates the G1Ҁ“S transition. We identify protein domains required for Trim71 association with Argonaute2, localization to P-bodies, and for repression of reporter messenger RNAs. Trim71 knockdown prolongs the G1 phase of the cell cycle and slows embryonic stem cell proliferation, a phenotype that was rescued by depletion of Cdkn1a. Thus, we demonstrate that Trim71 is a factor that facilitates the G1Ҁ“S transition to promote rapid embryonic stem cell self-renewal. Embryonic stem cells have a shortened cell cycle that allows for rapid proliferation, but the exact mechanisms are unclear. Here, a microRNA target, Trim71, is shown to inhibit the expression of a cyclin-dependent kinase inhibitor, thus enabling the G1Ҁ“S phase cell cycle transition in embryonic stem cells.
         datePublished:2012-06-26T00:00:00Z
         dateModified:2012-06-26T00:00:00Z
         pageStart:1
         pageEnd:10
         sameAs:https://doi.org/10.1038/ncomms1909
         keywords:
            Cell proliferation
            Gene regulation
            miRNAs
            Self-renewal
            Science
            Humanities and Social Sciences
            multidisciplinary
         image:
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Figa_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig1_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig2_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig3_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig4_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig5_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig6_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig7_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig8_HTML.jpg
         isPartOf:
            name:Nature Communications
            issn:
               2041-1723
            volumeNumber:3
            type:
               Periodical
               PublicationVolume
         publisher:
            name:Nature Publishing Group UK
            logo:
               url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
               type:ImageObject
            type:Organization
         author:
               name:Hao-Ming Chang
               affiliation:
                     name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
                     address:
                        name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
                        type:PostalAddress
                     type:Organization
               type:Person
               name:Natalia J. Martinez
               affiliation:
                     name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
                     address:
                        name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
                        type:PostalAddress
                     type:Organization
               type:Person
               name:James E. Thornton
               affiliation:
                     name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
                     address:
                        name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
                        type:PostalAddress
                     type:Organization
               type:Person
               name:John P. Hagan
               affiliation:
                     name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
                     address:
                        name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
                        type:PostalAddress
                     type:Organization
               type:Person
               name:Khang D. Nguyen
               affiliation:
                     name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
                     address:
                        name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
                        type:PostalAddress
                     type:Organization
               type:Person
               name:Richard I. Gregory
               affiliation:
                     name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
                     address:
                        name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
                        type:PostalAddress
                     type:Organization
               email:[email protected]
               type:Person
         isAccessibleForFree:
         hasPart:
            isAccessibleForFree:
            cssSelector:.main-content
            type:WebPageElement
         type:ScholarlyArticle
      context:https://schema.org
ScholarlyArticle:
      headline:Trim71 cooperates with microRNAs to repress Cdkn1a expression and promote embryonic stem cell proliferation
      description:Pluripotent embryonic stem cells have a shortened cell cycle that enables their rapid proliferation. The embryonic stem cell-specific miR-290 and miR-302 microRNA families promote proliferation whereas let-7 microRNAs inhibit self-renewal, and promote cell differentiation. Lin28 suppresses let-7 expression in embryonic stem cells. Here to gain further insight into mechanisms controlling embryonic stem cell self-renewal, we explore the molecular and cellular role of the let-7 target Trim71 (mLin41). We show that Trim71 associates with Argonaute2 and microRNAs, and represses expression of Cdkn1a, a cyclin-dependent kinase inhibitor that negatively regulates the G1Ҁ“S transition. We identify protein domains required for Trim71 association with Argonaute2, localization to P-bodies, and for repression of reporter messenger RNAs. Trim71 knockdown prolongs the G1 phase of the cell cycle and slows embryonic stem cell proliferation, a phenotype that was rescued by depletion of Cdkn1a. Thus, we demonstrate that Trim71 is a factor that facilitates the G1Ҁ“S transition to promote rapid embryonic stem cell self-renewal. Embryonic stem cells have a shortened cell cycle that allows for rapid proliferation, but the exact mechanisms are unclear. Here, a microRNA target, Trim71, is shown to inhibit the expression of a cyclin-dependent kinase inhibitor, thus enabling the G1Ҁ“S phase cell cycle transition in embryonic stem cells.
      datePublished:2012-06-26T00:00:00Z
      dateModified:2012-06-26T00:00:00Z
      pageStart:1
      pageEnd:10
      sameAs:https://doi.org/10.1038/ncomms1909
      keywords:
         Cell proliferation
         Gene regulation
         miRNAs
         Self-renewal
         Science
         Humanities and Social Sciences
         multidisciplinary
      image:
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Figa_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig1_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig2_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig3_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig4_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig5_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig6_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig7_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fncomms1909/MediaObjects/41467_2012_Article_BFncomms1909_Fig8_HTML.jpg
      isPartOf:
         name:Nature Communications
         issn:
            2041-1723
         volumeNumber:3
         type:
            Periodical
            PublicationVolume
      publisher:
         name:Nature Publishing Group UK
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Hao-Ming Chang
            affiliation:
                  name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
                  address:
                     name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Natalia J. Martinez
            affiliation:
                  name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
                  address:
                     name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:James E. Thornton
            affiliation:
                  name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
                  address:
                     name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:John P. Hagan
            affiliation:
                  name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
                  address:
                     name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Khang D. Nguyen
            affiliation:
                  name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
                  address:
                     name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Richard I. Gregory
            affiliation:
                  name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
                  address:
                     name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
["Periodical","PublicationVolume"]:
      name:Nature Communications
      issn:
         2041-1723
      volumeNumber:3
Organization:
      name:Nature Publishing Group UK
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
      address:
         name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
         type:PostalAddress
      name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
      address:
         name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
         type:PostalAddress
      name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
      address:
         name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
         type:PostalAddress
      name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
      address:
         name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
         type:PostalAddress
      name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
      address:
         name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
         type:PostalAddress
      name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
      address:
         name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:Hao-Ming Chang
      affiliation:
            name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
            address:
               name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
               type:PostalAddress
            type:Organization
      name:Natalia J. Martinez
      affiliation:
            name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
            address:
               name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
               type:PostalAddress
            type:Organization
      name:James E. Thornton
      affiliation:
            name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
            address:
               name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
               type:PostalAddress
            type:Organization
      name:John P. Hagan
      affiliation:
            name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
            address:
               name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
               type:PostalAddress
            type:Organization
      name:Khang D. Nguyen
      affiliation:
            name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
            address:
               name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
               type:PostalAddress
            type:Organization
      name:Richard I. Gregory
      affiliation:
            name:Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute
            address:
               name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
               type:PostalAddress
            type:Organization
      email:[email protected]
PostalAddress:
      name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
      name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
      name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
      name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
      name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
      name:Department of Biological Chemistry and Molecular Pharmacology, Stem Cell Program, Children's Hospital Boston, Harvard Medical School, Harvard Stem Cell Institute, Boston, USA
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

External Links {πŸ”—}(171)

Analytics and Tracking {πŸ“Š}

  • Google Tag Manager

Libraries {πŸ“š}

  • Foundation
  • Prism.js
  • Zoom.js

Emails and Hosting {βœ‰οΈ}

Mail Servers:

  • mxa-002c5801.gslb.pphosted.com
  • mxb-002c5801.gslb.pphosted.com

Name Servers:

  • pdns1.ultradns.net
  • pdns2.ultradns.net
  • pdns3.ultradns.org
  • pdns4.ultradns.org
  • pdns5.ultradns.info
  • pdns6.ultradns.co.uk

CDN Services {πŸ“¦}

  • Crossref

5.19s.