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Keywords {🔍}

cell, death, apoptosis, infection, nature, response, article, organisms, pathways, host, involved, nfκb, cas, google, scholar, apoptotic, molecular, caspase, biol, differ, related, individuals, parasite, proteins, cookies, content, differentiation, green, survival, protein, intracellular, activation, trigger, privacy, processing, caspases, components, altruistic, mechanisms, pathway, bacteria, domain, bcl, data, james, fungi, plants, role, cells, signaling,

Topics {✒️}

nature portfolio permissions reprints privacy policy nature 412 nature advertising proposed solutions social media author correspondence development trigger nf-κb-family activation potently activate nf-κb personal data data protection permissions nf-κb-family proteins infection-induced death response privacy nf-κb activation nf-κb pathways35 journals search log parasites' life cycle nf-κb pathways explore content altruistic cell death nuclear factor-κb undergo rapid death death effector domain programed cell death rapid cell death active cell death undergo cell death blocks cell death programmed cell death cell death differ european economic area low mutation rate exploit cellular resources nucleotide binding domain blocking systemic dissemination troublemaker daria capecedaniel nf-κb response accepting optional cookies caspase-activated interleukins anti-apoptotic actions inflammatory bowel disease genetic pathways leading single celled organisms caspase recruitment domain manage preferences

Questions {❓}

• Did mitochondria contribute the Bcl-2 family proteins to the apoptotic machinery of the eukaryotic cell as a mechanism for evasion of the infection-induced death response?
• So, if altruistic apoptosis evolved in protists, how could it be sustained and even favored?
• When did apoptosis evolve?

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