
NATURE . COM {
}
Title:
Regulation of breast cancer cell motility by insulin receptor substrate-2 (IRS-2) in metastatic variants of human breast cancer cell lines | Oncogene
Description:
Insulin-like growth factors (IGFs) regulate breast cancer cell proliferation, protect cells from apoptosis, and enhance metastasis. In this study, we examined the IGF signaling pathway in two breast cancer cell lines selected for metastatic behavior. LCC6 was selected for growth as an ascites tumor in athymic mice from parental MDA-MB-435 cells (435P). The MDA-231BO cell line was derived from osseous metastases that formed after intracardiac injection of the MDA-MB-231 cell line in athymic mice. Compared to the parental cell lines, IGF-I treatment enhanced IRS-2 phosphorylation over IRS-1 in the metastatic variants. IGF-I stimulated cell migration in the variant cells, but not in the parental cells. To determine the role for IRS-2 in IGF-mediated motility, we transfected MDA-231BO cells with an anti-sense IRS-2 construct. Transfected cells had decreased levels of IRS-2 with diminished IGF-mediated motility and anchorage independent growth when compared to control cells. However, adherence to fibronectin was enhanced in the transfected cells compared to MDA-231BO cells. Our data show that breast cancer cells selected for metastatic behavior in vivo have increased IRS-2 activation and signaling. In these cells, IGF-I enhances cell adhesion and motility suggesting that IRS-2 may mediate these aspects of the malignant phenotype.
Website Age:
30 years and 10 months (reg. 1994-08-11).
Matching Content Categories {π}
- Telecommunications
- Science
- Education
Content Management System {π}
What CMS is nature.com built with?
Custom-built
No common CMS systems were detected on Nature.com, and no known web development framework was identified.
Traffic Estimate {π}
What is the average monthly size of nature.com audience?
π Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.
check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush
How Does Nature.com Make Money? {πΈ}
Display Ads {π―}
The website utilizes display ads within its content to generate revenue. Check the next section for further revenue estimates.
Ads are managed by yourbow.com. Particular relationships are as follows:
Direct Advertisers (10)
google.com, pmc.com, doceree.com, yourbow.com, audienciad.com, onlinemediasolutions.com, advibe.media, aps.amazon.com, getmediamx.com, onomagic.comReseller Advertisers (38)
conversantmedia.com, rubiconproject.com, pubmatic.com, appnexus.com, openx.com, smartadserver.com, lijit.com, sharethrough.com, video.unrulymedia.com, google.com, yahoo.com, triplelift.com, onetag.com, sonobi.com, contextweb.com, 33across.com, indexexchange.com, media.net, themediagrid.com, adform.com, richaudience.com, sovrn.com, improvedigital.com, freewheel.tv, smaato.com, yieldmo.com, amxrtb.com, adyoulike.com, adpone.com, criteo.com, smilewanted.com, 152media.info, e-planning.net, smartyads.com, loopme.com, opera.com, mediafuse.com, betweendigital.comHow Much Does Nature.com Make? {π°}
Display Ads {π―}
$63,100 per month
According to our algorithms, Nature.com's monthly online income from display advertising ranges from $42,042 to $115,616.
Keywords {π}
cancer, cell, article, breast, nature, cells, biol, irs, yee, access, res, receptor, insulin, google, scholar, content, substrate, jackson, growth, chem, cas, cookies, oncogene, privacy, data, motility, metastatic, yoneda, open, journal, november, lines, insulinlike, parental, igfi, lee, texas, san, antonio, advertising, information, subscribe, variants, james, zhang, douglas, signaling, selected, mdamb, mdabo,
Topics {βοΈ}
nature portfolio permissions reprints privacy policy nature public health service immune prognostic index advertising social media author information authors glycolysis-related gene signature author correspondence mda-231bo cell line mda-mb-231 cell line diminished igf-mediated motility anti-sense irs-2 construct parental mda-mb-435 cells transfected mda-231bo cells springerlink instant access permissions insulin receptor substrate-2 insulin receptor substrate-1 insulin receptor substrate-4 personal data stimulated cell migration enhances cell adhesion data protection data show parental cell lines high-risk patients mda-231bo cells privacy mda-mb-435 parental issue learn igf-mediated motility breast cancer explore content subscription content drs robert clark anchorage independent growth european economic area institutional subscriptions read le marchand-brustel val toledo-loboluis accepting optional cookies journals search log de frias dv igf signaling pathway converting signaling dynamics growth factors manage preferences
Schema {πΊοΈ}
WebPage:
mainEntity:
headline:Regulation of breast cancer cell motility by insulin receptor substrate-2 (IRS-2) in metastatic variants of human breast cancer cell lines
description:Insulin-like growth factors (IGFs) regulate breast cancer cell proliferation, protect cells from apoptosis, and enhance metastasis. In this study, we examined the IGF signaling pathway in two breast cancer cell lines selected for metastatic behavior. LCC6 was selected for growth as an ascites tumor in athymic mice from parental MDA-MB-435 cells (435P). The MDA-231BO cell line was derived from osseous metastases that formed after intracardiac injection of the MDA-MB-231 cell line in athymic mice. Compared to the parental cell lines, IGF-I treatment enhanced IRS-2 phosphorylation over IRS-1 in the metastatic variants. IGF-I stimulated cell migration in the variant cells, but not in the parental cells. To determine the role for IRS-2 in IGF-mediated motility, we transfected MDA-231BO cells with an anti-sense IRS-2 construct. Transfected cells had decreased levels of IRS-2 with diminished IGF-mediated motility and anchorage independent growth when compared to control cells. However, adherence to fibronectin was enhanced in the transfected cells compared to MDA-231BO cells. Our data show that breast cancer cells selected for metastatic behavior in vivo have increased IRS-2 activation and signaling. In these cells, IGF-I enhances cell adhesion and motility suggesting that IRS-2 may mediate these aspects of the malignant phenotype.
datePublished:2001-11-16T00:00:00Z
dateModified:2001-11-16T00:00:00Z
pageStart:7318
pageEnd:7325
sameAs:https://doi.org/10.1038/sj.onc.1204920
keywords:
breast neoplasms
insulin-like growth factors
cell migration
insulin receptor substrate
adhesion
Medicine/Public Health
general
Internal Medicine
Cell Biology
Human Genetics
Oncology
Apoptosis
image:
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig1_HTML.jpg
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig2_HTML.jpg
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig3_HTML.jpg
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig4_HTML.jpg
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig5_HTML.jpg
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig6_HTML.jpg
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig7_HTML.jpg
isPartOf:
name:Oncogene
issn:
1476-5594
0950-9232
volumeNumber:20
type:
Periodical
PublicationVolume
publisher:
name:Nature Publishing Group UK
logo:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
type:ImageObject
type:Organization
author:
name:James G Jackson
affiliation:
name:University of Texas Health Science Center at San Antonio
address:
name:Department of Medicine, Division of Medical Oncology, University of Texas Health Science Center at San Antonio, San Antonio, USA
type:PostalAddress
type:Organization
type:Person
name:Xihong Zhang
affiliation:
name:Oncology, and Transplantation, University of Minnesota
address:
name:Department of Medicine, Division of Hematology, Oncology, and Transplantation, University of Minnesota, Minneapolis, USA
type:PostalAddress
type:Organization
type:Person
name:Toshiyuki Yoneda
affiliation:
name:University of Texas Health Science Center at San Antonio
address:
name:Department of Medicine, Division of Endocrinology, University of Texas Health Science Center at San Antonio, San Antonio, USA
type:PostalAddress
type:Organization
type:Person
name:Douglas Yee
affiliation:
name:Oncology, and Transplantation, University of Minnesota
address:
name:Department of Medicine, Division of Hematology, Oncology, and Transplantation, University of Minnesota, Minneapolis, USA
type:PostalAddress
type:Organization
email:[email protected]
type:Person
isAccessibleForFree:
hasPart:
isAccessibleForFree:
cssSelector:.main-content
type:WebPageElement
type:ScholarlyArticle
context:https://schema.org
ScholarlyArticle:
headline:Regulation of breast cancer cell motility by insulin receptor substrate-2 (IRS-2) in metastatic variants of human breast cancer cell lines
description:Insulin-like growth factors (IGFs) regulate breast cancer cell proliferation, protect cells from apoptosis, and enhance metastasis. In this study, we examined the IGF signaling pathway in two breast cancer cell lines selected for metastatic behavior. LCC6 was selected for growth as an ascites tumor in athymic mice from parental MDA-MB-435 cells (435P). The MDA-231BO cell line was derived from osseous metastases that formed after intracardiac injection of the MDA-MB-231 cell line in athymic mice. Compared to the parental cell lines, IGF-I treatment enhanced IRS-2 phosphorylation over IRS-1 in the metastatic variants. IGF-I stimulated cell migration in the variant cells, but not in the parental cells. To determine the role for IRS-2 in IGF-mediated motility, we transfected MDA-231BO cells with an anti-sense IRS-2 construct. Transfected cells had decreased levels of IRS-2 with diminished IGF-mediated motility and anchorage independent growth when compared to control cells. However, adherence to fibronectin was enhanced in the transfected cells compared to MDA-231BO cells. Our data show that breast cancer cells selected for metastatic behavior in vivo have increased IRS-2 activation and signaling. In these cells, IGF-I enhances cell adhesion and motility suggesting that IRS-2 may mediate these aspects of the malignant phenotype.
datePublished:2001-11-16T00:00:00Z
dateModified:2001-11-16T00:00:00Z
pageStart:7318
pageEnd:7325
sameAs:https://doi.org/10.1038/sj.onc.1204920
keywords:
breast neoplasms
insulin-like growth factors
cell migration
insulin receptor substrate
adhesion
Medicine/Public Health
general
Internal Medicine
Cell Biology
Human Genetics
Oncology
Apoptosis
image:
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig1_HTML.jpg
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig2_HTML.jpg
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig3_HTML.jpg
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig4_HTML.jpg
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig5_HTML.jpg
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig6_HTML.jpg
https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fsj.onc.1204920/MediaObjects/41388_2001_Article_BF1204920_Fig7_HTML.jpg
isPartOf:
name:Oncogene
issn:
1476-5594
0950-9232
volumeNumber:20
type:
Periodical
PublicationVolume
publisher:
name:Nature Publishing Group UK
logo:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
type:ImageObject
type:Organization
author:
name:James G Jackson
affiliation:
name:University of Texas Health Science Center at San Antonio
address:
name:Department of Medicine, Division of Medical Oncology, University of Texas Health Science Center at San Antonio, San Antonio, USA
type:PostalAddress
type:Organization
type:Person
name:Xihong Zhang
affiliation:
name:Oncology, and Transplantation, University of Minnesota
address:
name:Department of Medicine, Division of Hematology, Oncology, and Transplantation, University of Minnesota, Minneapolis, USA
type:PostalAddress
type:Organization
type:Person
name:Toshiyuki Yoneda
affiliation:
name:University of Texas Health Science Center at San Antonio
address:
name:Department of Medicine, Division of Endocrinology, University of Texas Health Science Center at San Antonio, San Antonio, USA
type:PostalAddress
type:Organization
type:Person
name:Douglas Yee
affiliation:
name:Oncology, and Transplantation, University of Minnesota
address:
name:Department of Medicine, Division of Hematology, Oncology, and Transplantation, University of Minnesota, Minneapolis, USA
type:PostalAddress
type:Organization
email:[email protected]
type:Person
isAccessibleForFree:
hasPart:
isAccessibleForFree:
cssSelector:.main-content
type:WebPageElement
["Periodical","PublicationVolume"]:
name:Oncogene
issn:
1476-5594
0950-9232
volumeNumber:20
Organization:
name:Nature Publishing Group UK
logo:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
type:ImageObject
name:University of Texas Health Science Center at San Antonio
address:
name:Department of Medicine, Division of Medical Oncology, University of Texas Health Science Center at San Antonio, San Antonio, USA
type:PostalAddress
name:Oncology, and Transplantation, University of Minnesota
address:
name:Department of Medicine, Division of Hematology, Oncology, and Transplantation, University of Minnesota, Minneapolis, USA
type:PostalAddress
name:University of Texas Health Science Center at San Antonio
address:
name:Department of Medicine, Division of Endocrinology, University of Texas Health Science Center at San Antonio, San Antonio, USA
type:PostalAddress
name:Oncology, and Transplantation, University of Minnesota
address:
name:Department of Medicine, Division of Hematology, Oncology, and Transplantation, University of Minnesota, Minneapolis, USA
type:PostalAddress
ImageObject:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
name:James G Jackson
affiliation:
name:University of Texas Health Science Center at San Antonio
address:
name:Department of Medicine, Division of Medical Oncology, University of Texas Health Science Center at San Antonio, San Antonio, USA
type:PostalAddress
type:Organization
name:Xihong Zhang
affiliation:
name:Oncology, and Transplantation, University of Minnesota
address:
name:Department of Medicine, Division of Hematology, Oncology, and Transplantation, University of Minnesota, Minneapolis, USA
type:PostalAddress
type:Organization
name:Toshiyuki Yoneda
affiliation:
name:University of Texas Health Science Center at San Antonio
address:
name:Department of Medicine, Division of Endocrinology, University of Texas Health Science Center at San Antonio, San Antonio, USA
type:PostalAddress
type:Organization
name:Douglas Yee
affiliation:
name:Oncology, and Transplantation, University of Minnesota
address:
name:Department of Medicine, Division of Hematology, Oncology, and Transplantation, University of Minnesota, Minneapolis, USA
type:PostalAddress
type:Organization
email:[email protected]
PostalAddress:
name:Department of Medicine, Division of Medical Oncology, University of Texas Health Science Center at San Antonio, San Antonio, USA
name:Department of Medicine, Division of Hematology, Oncology, and Transplantation, University of Minnesota, Minneapolis, USA
name:Department of Medicine, Division of Endocrinology, University of Texas Health Science Center at San Antonio, San Antonio, USA
name:Department of Medicine, Division of Hematology, Oncology, and Transplantation, University of Minnesota, Minneapolis, USA
WebPageElement:
isAccessibleForFree:
cssSelector:.main-content
External Links {π}(37)
- How much income does https://link.springer.com/article/10.1038/sj.onc.1204920?utm_source=nature&utm_medium=referral&utm_campaign=buyArticle have?
- How much does https://doi.org/10.1002%2F%28SICI%291097-0215%2819991112%2983%3A4%3C497%3A%3AAID-IJC11%3E3.0.CO%3B2-D pull in monthly?
- http://scholar.google.com/scholar_lookup?&title=Integrins%20and%20E%E2%80%90cadherin%20cooperate%20with%20IGF%E2%80%90I%20to%20induce%20migration%20of%20epithelial%20colonic%20cells&journal=International%20Journal%20of%20Cancer&doi=10.1002%2F%28SICI%291097-0215%2819991112%2983%3A4%3C497%3A%3AAID-IJC11%3E3.0.CO%3B2-D&volume=83&issue=4&pages=497-505&publication_year=1999&author=Andr%C3%A9%2CFr%C3%A9d%C3%A9ric&author=Rigot%2CV%C3%A9ronique&author=Thimonier%2CJean&author=Montixi%2CChristine&author=Parat%2CFabrice&author=Pommier%2CGilbert&author=Marvaldi%2CJacques&author=Luis%2CJos%C3%A9 income
- What are the total earnings of https://doi.org/10.1210%2Fendo.140.10.7027?
- How much does http://scholar.google.com/scholar_lookup?&title=Synthetic%20%CE%B1V%CE%B23%20Antagonists%20Inhibit%20Insulin-Like%20Growth%20Factor-I-Stimulated%20Smooth%20Muscle%20Cell%20Migration%20and%20Replication1&journal=Endocrinology&doi=10.1210%2Fendo.140.10.7027&volume=140&issue=10&pages=4616-4621&publication_year=1999&author=Clemmons%2CDavid%20R.&author=Horvitz%2CGayle&author=Engleman%2CWayne&author=Nichols%2CTim&author=Moralez%2CAnna&author=Nickols%2CG.%20Allen pull in?
- What's the monthly income of https://doi.org/10.1074%2Fjbc.273.16.9994?
- How much does http://scholar.google.com/scholar_lookup?&title=Insulin%20Receptor%20Substrate-1%20is%20the%20Predominant%20Signaling%20Molecule%20Activated%20by%20Insulin-like%20Growth%20Factor-I%2C%20Insulin%2C%20and%20Interleukin-4%20in%20Estrogen%20Receptor-positive%20Human%20Breast%20Cancer%20Cells&journal=Journal%20of%20Biological%20Chemistry&doi=10.1074%2Fjbc.273.16.9994&volume=273&issue=16&pages=9994-10003&publication_year=1998&author=Jackson%2CJames%20G.&author=White%2CMorris%20F.&author=Yee%2CDouglas make?
- What's the total monthly financial gain of https://doi.org/10.1016%2FS0092-8674%2800%2981280-5?
- http://scholar.google.com/scholar_lookup?&title=Cell%20Migration%3A%20A%20Physically%20Integrated%20Molecular%20Process&journal=Cell&doi=10.1016%2FS0092-8674%2800%2981280-5&volume=84&issue=3&pages=359-369&publication_year=1996&author=Lauffenburger%2CDouglas%20A&author=Horwitz%2CAlan%20F's revenue stream
- How much does https://doi.org/10.1074%2Fjbc.M006162200 net monthly?
- Check the income stats for http://scholar.google.com/scholar_lookup?&title=Cell%20Adhesion%20and%20Focal%20Adhesion%20Kinase%20Regulate%20Insulin%20Receptor%20Substrate-1%20Expression&journal=Journal%20of%20Biological%20Chemistry&doi=10.1074%2Fjbc.M006162200&volume=275&issue=49&pages=38371-38377&publication_year=2000&author=Lebrun%2CPatricia&author=Baron%2CV%C3%A9ronique&author=Hauck%2CChristof%20R.&author=Schlaepfer%2CDavid%20D.&author=Van%20Obberghen%2CEmmanuel
- How much money does https://doi.org/10.1074%2Fjbc.274.32.22401 generate?
- How much does http://scholar.google.com/scholar_lookup?&title=Extracellular%20Matrix%20Selectively%20Modulates%20the%20Response%20of%20Mammary%20Epithelial%20Cells%20to%20Different%20Soluble%20Signaling%20Ligands&journal=Journal%20of%20Biological%20Chemistry&doi=10.1074%2Fjbc.274.32.22401&volume=274&issue=32&pages=22401-22408&publication_year=1999&author=Lee%2CYi-Ju&author=Streuli%2CCharles%20H. rake in every month?
- How much does https://doi.org/10.1038%2Fsj.onc.1204112 make?
- Revenue of http://scholar.google.com/scholar_lookup?&title=Mechanisms%20of%20regulation%20of%20cell%20adhesion%20and%20motility%20by%20insulin%20receptor%20substrate-1%20in%20prostate%20cancer%20cells&journal=Oncogene&doi=10.1038%2Fsj.onc.1204112&volume=20&issue=4&pages=490-500&publication_year=2001&author=Reiss%2CKrzysztof&author=Wang%2CJin-Ying&author=Romano%2CGaetano&author=Tu%2CXiao&author=Peruzzi%2CFrancesca&author=Baserga%2CRenato
- How much does https://doi.org/10.1126%2Fscience.7527156 rake in every month?
- Learn about the earnings of http://scholar.google.com/scholar_lookup?&title=Association%20of%20insulin%20receptor%20substrate-1%20with%20integrins&journal=Science&doi=10.1126%2Fscience.7527156&volume=266&issue=5190&pages=1576-1578&publication_year=1994&author=Vuori%2CK&author=Ruoslahti%2CE
- What are the total earnings of https://citation-needed.springer.com/v2/references/10.1038/sj.onc.1204920?format=refman&flavour=references?
- How much profit does https://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=search&term=James%20G%20Jackson generate?
- What's the total monthly financial gain of https://scholar.google.co.uk/scholar?as_q=&num=10&btnG=Search+Scholar&as_epq=&as_oq=&as_eq=&as_occt=any&as_sauthors=%22James%20G%20Jackson%22&as_publication=&as_ylo=&as_yhi=&as_allsubj=all&hl=en?
- Earnings of https://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=search&term=Xihong%20Zhang
- https://scholar.google.co.uk/scholar?as_q=&num=10&btnG=Search+Scholar&as_epq=&as_oq=&as_eq=&as_occt=any&as_sauthors=%22Xihong%20Zhang%22&as_publication=&as_ylo=&as_yhi=&as_allsubj=all&hl=en income
- How much does https://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=search&term=Toshiyuki%20Yoneda rake in every month?
- What's the total monthly financial gain of https://scholar.google.co.uk/scholar?as_q=&num=10&btnG=Search+Scholar&as_epq=&as_oq=&as_eq=&as_occt=any&as_sauthors=%22Toshiyuki%20Yoneda%22&as_publication=&as_ylo=&as_yhi=&as_allsubj=all&hl=en?
- How much profit does https://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=search&term=Douglas%20Yee generate?
- How much money does https://scholar.google.co.uk/scholar?as_q=&num=10&btnG=Search+Scholar&as_epq=&as_oq=&as_eq=&as_occt=any&as_sauthors=%22Douglas%20Yee%22&as_publication=&as_ylo=&as_yhi=&as_allsubj=all&hl=en make?
- What's the financial outcome of https://s100.copyright.com/AppDispatchServlet?title=Regulation%20of%20breast%20cancer%20cell%20motility%20by%20insulin%20receptor%20substrate-2%20%28IRS-2%29%20in%20metastatic%20variants%20of%20human%20breast%20cancer%20cell%20lines&author=James%20G%20Jackson%20et%20al&contentID=10.1038%2Fsj.onc.1204920©right=Macmillan%20Publishers%20Limited&publication=0950-9232&publicationDate=2001-11-16&publisherName=SpringerNature&orderBeanReset=true?
- What is the monthly revenue of https://citation-needed.springer.com/v2/references/10.1038/sj.onc.1204920?format=refman&flavour=citation?
- What is the earnings of https://doi.org/10.1007/s12325-022-02330-y?
- What's the financial gain of https://doi.org/10.1038/s41416-020-01094-y?
- What is the monthly revenue of https://doi.org/10.1186/s12885-018-4950-0?
- What's the financial intake of https://doi.org/10.1007/s10735-017-9745-0?
- What's the income of https://doi.org/10.1007/s12672-018-0343-8?
- What are the total earnings of https://www.protocols.io/?
- Financial intake of https://www.natureindex.com/
- Explore the financials of http://www.naturechina.com
- Check the income stats for https://www.natureasia.com/ja-jp
Analytics and Tracking {π}
- Google Tag Manager
Libraries {π}
- Prism.js
- Zoom.js
Emails and Hosting {βοΈ}
Mail Servers:
- mxa-002c5801.gslb.pphosted.com
- mxb-002c5801.gslb.pphosted.com
Name Servers:
- pdns1.ultradns.net
- pdns2.ultradns.net
- pdns3.ultradns.org
- pdns4.ultradns.org
- pdns5.ultradns.info
- pdns6.ultradns.co.uk
CDN Services {π¦}
- Crossref