Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/9746332/.

Title:
Mitochondrial function as a determinant of recovery or death in cell response to injury - PubMed
Description:
Many pathological conditions can be the cause or the consequence of mitochondrial dysfunction. For instance anoxia, which is initiated by a critical reduction of oxygen availability for mitochondrial oxidations, is followed by a wide variety of mitochondrial alterations. A crucial role in the evolut …
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {📚}

  • Telecommunications
  • Fitness & Wellness
  • Insurance

Content Management System {📝}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {💸}

We can't figure out the monetization strategy.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Pubmed.ncbi.nlm.nih.gov might be cashing in, but we can't detect the method they're using.

Keywords {🔍}

mitochondrial, pmid, cell, pubmed, doi, biochem, mol, jul, injury, permeability, articles, mesh, cellular, oxidative, transition, pore, review, reperfusion, pmc, main, ncbi, abstract, similar, cited, references, resources, function, dysfunction, alterations, role, mitochondria, damage, phosphorylation, heart, opening, ischemia, potential, epub, channels, free, article, physiology, physiopathology, hhs, page, content, homepage, navigation, search, publication,

Topics {✒️}

main page content ischemia-reperfusion injury ischemia/reperfusion-induced damage oxidative phosphorylation similar articles cited ischemic myocardium early ischemia-induced alterations outer mitochondrial membrane permeability transition pore turn mitochondria functional mitochondria intact heart ageing heart cardiac ischemic injury literature sources mesh keyword cell injury oxidative stress massive mitochondrial uptake mol cell cardiol mol cell biochem cell mol neurobiol exacerbate cell damage simulated ischemia simulated reperfusion united states government proton electrochemical gradient general consensus emerges altered energy transfer isolated rat hearts injury intracellular ca2+ overload ischemia reperfusion funding linkout reduced atp availability cell response cardiovasc res cited mitochondrial alterations promote opening cardiac muscle 9746332 abstract abstract mitochondrial function mitochondrial oxidations mitochondrial point 7512654 mitochondrial function mitochondrial contribution cellular damage

Questions {❓}

  • Early ischemia-induced alterations of the outer mitochondrial membrane and the intermembrane space: a potential cause for altered energy transfer in cardiac muscle?
  • Oxidative stress in phenylketonuria: what is the evidence?

External Links {🔗}(48)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • jQuery

CDN Services {📦}

  • Ncbi

2.92s.