Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/23983249/.

Title:
Depressing mitochondria-reticulum interactions protects cardiomyocytes from lethal hypoxia-reoxygenation injury - PubMed
Description:
Our data (1) point to a new role of CypD at the ER-mitochondria interface and (2) suggest that decreasing ER-mitochondria interaction at reperfusion can protect cardiomyocytes against lethal reperfusion injury through the reduction of mitochondrial Ca(2+) overload via the CypD/VDAC1/Grp75/IP3R1 comp …
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {📚}

  • Science
  • Fitness & Wellness
  • Education

Content Management System {📝}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {💸}

We find it hard to spot revenue streams.

Not all websites focus on profit; some are designed to educate, connect people, or share useful tools. People create websites for numerous reasons. And this could be one such example. Pubmed.ncbi.nlm.nih.gov might be cashing in, but we can't detect the method they're using.

Keywords {🔍}

doi, pmid, physiology, injury, cypd, epub, hypoxiareoxygenation, cell, zhang, cardiomyocytes, mitochondria, complex, pmc, transfer, reticulum, mitochondrial, interaction, calcium, wang, free, article, proteins, similar, articles, mesh, ipr, protein, mice, ermitochondria, reperfusion, liu, cyclophilins, myocytes, cardiac, main, content, ncbi, cited, resources, protects, teixeira, ovize, endoplasmic, role, death, inhibition, cells, pubmed, mar, membrane,

Topics {✒️}

main page content similar articles cited mitochondria-endoplasmic reticulum interactions mouse vdac1 protein cypd/vdac1/grp75/ip3r1 complex vdac1/grp75/ip3r1 complex mitochondria links alterations inbred c57bl mice er membrane integrity endoplasmic reticulum adult mice cardiomyocytes decreasing er-mitochondria interaction lethal reperfusion injury similar effect mol cell cardiol literature sources cell death meet cell death dis ischemic postconditioning protects ischemia/reperfusion injury ischemia-reperfusion injury hypoxia-reoxygenation injury hypoxia/reoxygenation injury mesh keyword mitochondrial chaperone cyclophilin cell calcium adult cardiomyocytes decreased united states government resources circulation mfn2 similarly reduced hepatic insulin resistance front cardiovasc med attenuated mitochondrial ca local calcium transfer pierre-alain thiebaut protected cells er-mitochondria interface er-mitochondria interaction sarcoplasmic reticulum reperfusion injury ip3r1 complex cited hypoxia-reoxygenation human services mitochondrial matrix pubmed wordmark pubmed logo mitochondrial ca calcium transfer heart

External Links {🔗}(74)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • jQuery

CDN Services {📦}

  • Ncbi

3.35s.