Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/23744057/.

Title:
Endoplasmic reticulum stress contributes to heart protection induced by cyclophilin D inhibition - PubMed
Description:
Preventing cyclophilin D (cypD) translocation to the inner mitochondrial membrane can limit lethal reperfusion injury through the inhibition of the opening of the mitochondrial permeability transition pore. Inhibition or loss of function of cypD may also result into an endoplasmic reticulum (ER) str …
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {📚}

  • Health & Fitness
  • Fitness & Wellness
  • Pets

Content Management System {📝}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {💸}

We can't see how the site brings in money.

Not all websites focus on profit; some are designed to educate, connect people, or share useful tools. People create websites for numerous reasons. And this could be one such example. Pubmed.ncbi.nlm.nih.gov might be making money, but it's not detectable how they're doing it.

Keywords {🔍}

doi, stress, pmid, endoplasmic, reticulum, mice, epub, cypd, mitochondrial, injury, pmc, cyclophilin, inhibition, cell, free, article, myocardial, ovize, function, ischemiareperfusion, mol, cyclophilins, similar, articles, mesh, cardiol, heart, reperfusion, permeability, transition, infarct, size, cyclosporine, acid, main, ncbi, cited, information, resources, induced, lethal, pore, role, cypdko, protein, bip, pubmed, cardiomyocytes, teixeira, abrial,

Topics {✒️}

myocardial ischemia-reperfusion injury endoplasmic reticulum stress lethal ischemia-reperfusion injury main page content similar articles cited endoplasmic reticulum lethal hypoxia-reoxygenation injury male mice heart protection induced cypd-deficient mice displayed cardiac ischemia-reperfusion cypd-ko mice exhibited mesh keyword prolonged ischemia-reperfusion ischemia-induced facial palsy health wild-type mice inbred c57bl mice er stress induced er stress markers er stress proteins cypd deficiency heart mouse ppif protein neonatal rat cardiomyocytes reperfusion phase bip similar results treated mice isoflurane-induced preconditioning mol cell cardiol er stress cell mol neurobiol alter cell survival united states government triphenyltetrazolium chloride staining decreased infarct size da silva cc hexosamine biosynthetic pathway mitochondrial membrane left ventricular weight mitochondrial cyclophilin potential therapeutic target cypd function plays inhibit cypd function 4-phenylbutyric acid cited pubmed wordmark pubmed logo stress

Questions {❓}

  • Mitochondrial permeability transition in cardiac ischemia-reperfusion: whether cyclophilin D is a viable target for cardioprotection?

External Links {🔗}(61)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • jQuery

CDN Services {📦}

  • Ncbi

3.03s.