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Title:
Is p53 the long-sought molecular trigger for cyclophilin D-regulated mitochondrial permeability transition pore formation and necrosis? - PubMed
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Is p53 the long-sought molecular trigger for cyclophilin D-regulated mitochondrial permeability transition pore formation and necrosis?
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Keywords {π}
pmid, doi, mitochondrial, permeability, transition, pore, pmc, cell, free, article, review, comment, articles, res, oct, epub, main, ncbi, similar, cited, resources, circresaha, cyclophilin, pubmed, death, nov, yang, hhs, page, content, homepage, navigation, search, full, text, publication, types, linkout, circ, trigger, necrosis, abstract, disclaimer, increased, cyclophilind, vulnerability, mar, redox, characterization, human,
Topics {βοΈ}
main page content cyclophilin d-dependent control mitochondrial research long-sought molecular trigger 19094991 redox characterization permeability transition pore mitochondrial atp synthase necrotic cell death united states government novartis found symp compensated ventricular hypertrophy arch biochem biophys oxygen glucose deprivation/ oxygen glucose deprivation biochim biophys acta mol cell cardiol stress-induced opening similar articles cited /p53 signaling axis cell death increased vulnerability human cyclophilin targeting cyclophilin similar articles circ res hhs trigger necrosis cancer res neurochem res pubmed wordmark pubmed logo official website jason karchΒ 1 p53 opens vaseva av tsirka se moll um honokiol induces 17510419 increased expression intramitochondrial translocation cyclophilin young nt bourcier-lucas deschepper cf dietz kj tang dr oxygenation-induced zhao lp cardiac pathophysiology cardiovasc dis
Questions {β}
- Is p53 the long-sought molecular trigger for cyclophilin D-regulated mitochondrial permeability transition pore formation and necrosis?
- Redox characterization of human cyclophilin D: identification of a new mammalian mitochondrial redox sensor?
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