PUBMED . NCBI . NLM . NIH . GOV {
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Title:
Cysteine 203 of cyclophilin D is critical for cyclophilin D activation of the mitochondrial permeability transition pore - PubMed
Description:
The mitochondrial permeability transition pore (mPTP) opening plays a critical role in mediating cell death during ischemia/reperfusion (I/R) injury. Our previous studies have shown that cysteine 203 of cyclophilin D (CypD), a critical mPTP mediator, undergoes protein S-nitrosylation (SNO). To inves …
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Keywords {🔍}
mitochondrial, mptp, doi, cypd, permeability, pmid, epub, transition, pore, pmc, pubmed, cyclophilin, cscypd, free, article, opening, mice, nih, metabolism, states, cysteine, activation, figure, review, protein, mefs, hhsunited, articles, mesh, nov, role, mouse, mitochondria, cyclophilins, physiol, main, ncbi, figures, similar, cited, references, grants, resources, critical, cell, ischemiareperfusion, injury, fibroblasts, nitric, oxide,
Topics {✒️}
united states government substances peptidyl-prolyl isomerase hhs main page content similar articles cited funding linkout nitric oxide donor permeability transition pore hepatic ischemia/reperfusion injury cyclophilin d-dependent control undergoes protein s-nitrosylation mitochondrial calcein fluorescence articles references griffiths c203s-cypd mutation mitochondria isolated mice expressing c203s-cypd wt cypd-reconstituted mice medgen protein protein redox stress-induced activation mitochondria mitochondrial ca2+ cyclophilins block opening c203s-cypd reconstituted mefs mptp mouse model pubchem substance grants mesh keyword ischemia/reperfusion mediating cell death 243469 abstract antioxid redox signal mutation ms/ms spectra curr pharm des post-translational modifications biochim biophys acta proximal tubule cyclophilin mediates kidney fibrogenesis blood flow metab c203s-cypd vector c203s-cypd reduces mefs exhibited significantly resources mutated c203s-cypd figures suggesting substantial activation cited attenuated mptp opening protein signaling critical mptp mediator physiol renal physiol
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