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Title:
Mitochondrial permeability transition in Ca(2+)-dependent apoptosis and necrosis - PubMed
Description:
A variety of stimuli utilize an increase of cytosolic free Ca(2+) concentration as a second messenger to transmit signals, through Ca(2+) release from the endoplasmic reticulum or opening of plasma membrane Ca(2+) channels. Mitochondria contribute to the tight spatiotemporal control of this process …
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Keywords {🔍}
mitochondrial, doi, pmid, cell, free, metabolism, permeability, review, calcium, transition, death, pmc, epub, opening, membrane, article, diseases, apoptosis, pore, ptp, main, articles, cited, mesh, release, matrix, pathology, ncbi, similar, resources, jceca, concentration, endoplasmic, reticulum, proteins, signalling, disease, pubmed, jul, liver, aug, hhs, page, content, homepage, navigation, search, full, text, abstract,
Topics {✒️}
permeability transition pore mitochondrial permeability transition chronic liver injury endoplasmic reticulum apoptogenic proteins disease pathogenesis necrosis andrea rasola 1 cell calcium similar articles cited mitochondria contribute calcium plasma membrane ca membrane channel membrane unfolding mitochondrial ca2+ uptake mesh keyword mitochondrial free ca mediating cell death main page content cited mitochondrial ca cell death mitochondrial connection mitochondrial roles mitochondrial channels necrotic death signals dependent apoptosis apoptosis main biological routines review 18193649 review 17294079 review cell microglia-induced neuroinflammation united states government 007 abstract tight spatiotemporal control reactive oxygen species traumatic brain injury viral phase separation post-acute sequelae febs open bio persistent ptp opening funding linkout cytosolic free ca biochem res int endogenous ca2+ inducing opening pubmed wordmark pubmed logo
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