Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/20080742/.

Title:
Activation of mitochondrial ERK protects cancer cells from death through inhibition of the permeability transition - PubMed
Description:
We studied human cancer cell models in which we detected constitutive activation of ERK. A fraction of active ERK was found to be located in mitochondria in RWPE-2 cells, obtained by v-Ki-Ras transformation of the epithelial prostate RWPE-1 cell line; in metastatic prostate cancer DU145 cells; and i โ€ฆ
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {๐Ÿ“š}

  • Science
  • Telecommunications
  • Law & Government

Content Management System {๐Ÿ“}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {๐Ÿ“ˆ}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

๐ŸŒ  Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {๐Ÿ’ธ}

We can't tell how the site generates income.

Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Pubmed.ncbi.nlm.nih.gov might be cashing in, but we can't detect the method they're using.

Keywords {๐Ÿ”}

mitochondrial, doi, cell, cells, pmid, pmc, erk, permeability, transition, pore, pubmed, cancer, mitochondria, free, article, death, bernardi, epub, activation, inhibition, rasola, rwpe, tumor, review, articles, sci, dec, sciacovelli, resistance, ptp, drug, cyclophilin, fig, metabolism, main, ncbi, conflict, interest, similar, cited, references, mesh, resources, prostate, acid, feb, map, hhs, page, content,

Topics {โœ’๏ธ}

amino acid metabolism mitochondrial permeability transition permeability transition pore mitochondria signal transduction mitochondrial respiratory complex main page content tumor cell death mitochondrial dna mitochondrial dysfunction mitochondrial abnormalities detected constitutive activation drug resistance drug pd98059 pore regulator cyclophilin tumor metastasis raf/mek/erk pathway pore desensitization similar articles cited activation v-ki-ras transformation articles references dhillon cell death induced cell death dis authors declare prostate cancer progression gsk-3 inhibition protected osteosarcoma saos-2 cells transformed rwpe-1 cells united states government biochim biophys acta sirt3 deficient hearts nat rev cancer health cell death 0912742107 abstract bh3 mimetic em20-25 cardiac ischemia-reperfusion parodi-rullรกn rm interest statement 22402226 chemotherapeutic induction cell growth oxidative damage conflict rwpe-2 cells pubmed schubbert pubmed show pubmed wordmark pubmed logo active erk

Questions {โ“}

  • Cyclophilin D in Mitochondrial Dysfunction: A Key Player in Neurodegeneration?
  • Mitochondrial permeability transition in cardiac ischemia-reperfusion: whether cyclophilin D is a viable target for cardioprotection?

External Links {๐Ÿ”—}(58)

Analytics and Tracking {๐Ÿ“Š}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {๐Ÿ“š}

  • AOS
  • Clipboard.js
  • jQuery
  • PhotoSwipe

CDN Services {๐Ÿ“ฆ}

  • Ncbi

3.17s.