Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/20026007/.

Title:
Knockdown of F1 epsilon subunit decreases mitochondrial content of ATP synthase and leads to accumulation of subunit c - PubMed
Description:
The subunit epsilon of mitochondrial ATP synthase is the only F1 subunit without a homolog in bacteria and chloroplasts and represents the least characterized F1 subunit of the mammalian enzyme. Silencing of the ATP5E gene in HEK293 cells resulted in downregulation of the activity and content of the โ€ฆ
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {๐Ÿ“š}

  • Science
  • Law & Government
  • Telecommunications

Content Management System {๐Ÿ“}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {๐Ÿ“ˆ}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

๐ŸŒ  Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,009,998 visitors per month in the current month.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {๐Ÿ’ธ}

We're unsure how the site profits.

Not all websites focus on profit; some are designed to educate, connect people, or share useful tools. People create websites for numerous reasons. And this could be one such example. Pubmed.ncbi.nlm.nih.gov has a secret sauce for making money, but we can't detect it yet.

Keywords {๐Ÿ”}

mitochondrial, subunit, synthase, doi, atp, pmid, epub, subunits, content, epsilon, free, article, gene, protein, pmc, complex, sep, protontranslocating, atpases, proteins, articles, mesh, jbbabio, assembly, main, ncbi, similar, cited, related, resources, biochim, biophys, acta, kaplanovรก, atpe, cells, role, pubmed, mol, jun, human, review, cell, genetics, rna, pubchem, hhs, page, homepage, navigation,

Topics {โœ’๏ธ}

hek293 cells resulted medgen protein protein knockdown mitochondrial atp synthase similar articles cited selective mitochondrial inhibition main page content neuroblastoma cells mesh keyword mitochondrial respiration nuclear atp5e gene f1fo-atp synthase nanomotor f1 subunits alpha atp5e gene f1 subunits related ฮฑ-proteobacteria fo subunits atp synthase united states government f1 epsilon subunit garcรญa-trejo jj modulating ampk signaling poor prognostic marker colorectal cancer patients f1-c-ring mol cell cardiol adp-stimulated respiration hum mol genet di rago jp characterized f1 subunit cell mol med human pathogenic mutation cited subunit epsilon epsilon subunit pubmed wordmark pubmed logo decreased content f1 subunit mesenchymal transition human services hhs hydrophobic subunit cell survival front genet clin med di lisa official website zdenek drahota mammalian enzyme

External Links {๐Ÿ”—}(54)

Analytics and Tracking {๐Ÿ“Š}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {๐Ÿ“š}

  • Clipboard.js
  • jQuery

CDN Services {๐Ÿ“ฆ}

  • Ncbi

3.47s.