Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/19447775/.

Title:
Regulation and pharmacology of the mitochondrial permeability transition pore - PubMed
Description:
The
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {📚}

  • Science
  • Fitness & Wellness
  • Law & Government

Content Management System {📝}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {💸}

We don't see any clear sign of profit-making.

While profit motivates many websites, others exist to inspire, entertain, or provide valuable resources. Websites have a variety of goals. And this might be one of them. Pubmed.ncbi.nlm.nih.gov could have a money-making trick up its sleeve, but it's undetectable for now.

Keywords {🔍}

mitochondrial, pmc, doi, transition, pmid, membrane, metabolism, permeability, pubmed, free, article, review, pore, mptp, epub, regulation, proteins, mitochondria, transport, articles, mesh, adenine, nucleotide, cyclophilin, figure, biochem, biophys, main, ncbi, similar, cited, references, substances, information, resources, research, death, kinase, drug, translocator, voltagedependent, anion, channels, molkentin, ischemiareperfusion, dec, cell, apr, heart, haworth,

Topics {✒️}

mitochondrial membrane permeability voltage-dependent anion channels ca2+-induced membrane transition 'mitochondrial permeability transition' main page content similar articles cited membrane channel activity pore-forming core component pubchem substance grants ischemia/reperfusion injury articles references mitchell 1093/cvr/cvp151 abstract ser/thr protein phosphatases drug design aged heart cyclophilin d-overlapping pathways mitochondrial dysfunction mitochondrial mitochondria united states government ischemia-reperfusion phenomena literature sources mitochondrial mesh keyword small substances sudden induced change kinase signalling ca2+ trigger site transitional ca2+ release arch biochem biophys extensive research efforts funding linkout necrotic cell death vidal-puig aj potential mechanisms mptp remain unidentified pharmacology cell death regulation cited brand md pubmed show pubmed wordmark pubmed logo research interest hunter dr hhs 1093/cvr/cvp151 core elements sci adv uncoupling protein 1 calcium modulation

Questions {❓}

  • Mitochondrial dysfunction and mitophagy: the beginning and end to diabetic nephropathy?
  • Regulation of necrotic cell death: p53, PARP1 and cyclophilin D-overlapping pathways of regulated necrosis?

External Links {🔗}(70)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • jQuery
  • PhotoSwipe

CDN Services {📦}

  • Ncbi

4.11s.