Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/17417626/.

Title:
Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death - PubMed
Description:
Mitochondria are critically involved in necrotic cell death induced by Ca(2+) overload, hypoxia and oxidative damage. The mitochondrial permeability transition (MPT) pore - a protein complex that spans both the outer and inner mitochondrial membranes - is considered the mediator of this event and ha …
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {📚}

  • Science
  • Fitness & Wellness
  • Telecommunications

Content Management System {📝}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {💸}

We can't tell how the site generates income.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Pubmed.ncbi.nlm.nih.gov might have a hidden revenue stream, but it's not something we can detect.

Keywords {🔍}

cell, mitochondrial, vdac, metabolism, death, doi, voltagedependent, anion, pmid, permeability, pmc, transition, pubmed, mitochondria, epub, free, article, channel, states, pore, protein, membrane, role, mouse, nih, proteins, biol, channels, mpt, figure, apoptosis, articles, mesh, cells, kroemer, atp, research, rna, hlhlnhlbi, hhsunited, main, ncbi, abstract, similar, cited, references, related, grants, resources, nat,

Topics {✒️}

voltage-dependent anion channels united states government voltage-dependent anion channel mitochondrial-dependent cell death oxidative stress-induced mpt oxidative-stress-induced mpt pubmed show mitochondrial permeability transition hhs main page content funding linkout mitochondrial membranes fo atp synthase f1fo atp synthase metabolism vdac1-vdac3-null mice exhibited pro-apoptotic ca2+ signals anti-apoptotic bcl-xl adenine-nucleotide translocase extramural research support mitochondrial apoptosis mouse vdac3 protein mouse vdac2 protein mesh keyword related bcl-2 mitochondrial adp outer membrane oxidative damage exhibited equivalent cytochrome membrane calcium biomedical research pore pore biochim biophys acta van vliet ar la rovere rm mammalian vdac genes nat cell biol rat liver mitochondria fibroblasts lacking vdac1 similar articles cited � comment critical control points vdac-deficient mitochondria mitochondria-mediated neurodegeneration cell death similar articles role vdac1-vdac2-vdac3 wild-type mitochondria

External Links {🔗}(93)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • jQuery
  • PhotoSwipe

CDN Services {📦}

  • Ncbi

3.25s.