Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/16839884/.

Title:
Mitochondrial rhomboid PARL regulates cytochrome c release during apoptosis via OPA1-dependent cristae remodeling - PubMed
Description:
Rhomboids, evolutionarily conserved integral membrane proteases, participate in crucial signaling pathways. Presenilin-associated rhomboid-like (PARL) is an inner mitochondrial membrane rhomboid of unknown function, whose yeast ortholog is involved in mitochondrial fusion. Parl-/- mice display norma …
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {πŸ“š}

  • Science
  • Education
  • Fitness & Wellness

Content Management System {πŸ“}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,003 visitors per month in the current month.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {πŸ’Έ}

We see no obvious way the site makes money.

Some websites aren't about earning revenue; they're built to connect communities or raise awareness. There are numerous motivations behind creating websites. This might be one of them. Pubmed.ncbi.nlm.nih.gov might be making money, but it's not detectable how they're doing it.

Keywords {πŸ”}

mitochondrial, doi, parl, pmid, opa, apoptosis, free, article, pmc, metabolism, cell, jul, protein, mitochondria, genetics, jcell, scorrano, epub, display, articles, cited, mesh, mice, death, review, proteins, main, ncbi, similar, resources, rhomboid, cytochrome, cristae, remodeling, membrane, cells, apoptotic, pubmed, biophys, oct, research, gtp, phosphohydrolases, metalloproteases, mouse, gene, hhs, page, content, access,

Topics {βœ’οΈ}

mitochondria-shaping proteins main page content mitochondrial membrane rhomboid metalloproteases parl protein / metabolism cultured cytochromes mitochondrial morphology inbred c57bl mice mitochondria mitochondrial fusion mitochondrial adaptation mitochondrial injury postnatal growth restriction rhomboid serine proteases martins de brito mesh keyword human parl protein opa1 specifically targeted united states government luciano d'adamio rudi d'hooge crucial signaling pathways sanjuΓ‘n szklarz lk novartis found symp higd-1a interacts medgen nucleotide omim biochim biophys acta resources cell funding linkout similar articles cited de strooper cell death differ intramembrane proteolysis protein protein biomedical research similar articles regulation opa1 processing hhs cachectic death atrophy grants human services increased apoptosis controlling apoptosis pubmed wordmark pubmed logo official website dieter hartmann veronica costa

External Links {πŸ”—}(70)

Analytics and Tracking {πŸ“Š}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {πŸ“š}

  • Clipboard.js
  • jQuery

CDN Services {πŸ“¦}

  • Ncbi

4.37s.