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Title:
Postconditioning inhibits mitochondrial permeability transition - PubMed
Description:
Postconditioning inhibits opening of the mPTP and provides a powerful antiischemic protection.
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27 years and 8 months (reg. 1997-10-02).
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Keywords {🔍}
doi, postconditioning, mitochondrial, pmid, reperfusion, permeability, transition, myocardial, ischemia, physiology, injury, epub, opening, pore, proteins, effects, drug, ion, mptp, minute, abstract, articles, mesh, circulation, inhibits, argaud, rabbits, preconditioning, article, oct, feb, pmc, map, kinases, transport, protooncogene, main, ncbi, similar, cited, resources, jan, gateauroesch, ovize, infarct, size, minutes, control, nim, mitochondria,
Topics {✒️}
myocardial reperfusion injury myocardial ischemia/reperfusion mitochondrial proteins mitochondrial permeability transition lethal reperfusion injury time control hearts underwent anti-apoptotic effects heart fail rev 001 versus control mitochondrial dna mitochondrial stat3 main page content ischemia performed prolonged ischemia 30-minute ischemia cyclosporine cyclosporine ischemia rabbits reperfusion performed reperfusion mitochondria ca2+-induced mptp opening similar articles cited powerful antiischemic protection literature sources postconditioning inhibits opening mesh keyword coronary occlusion modulate mptp opening reduce infarct size risk signalling pathway ca2+ load required mol cell cardiol united states government med sci monit molecular cardioprotective responses trends cardiovasc med basic res cardiol risk region myocardium author reply e442 isoflurane-induced odile gateau-roesch infarct size preconditioning consisted ischemic preconditioning significantly higher risk region abstract
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