Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/12881569/.

Title:
VDAC2 inhibits BAK activation and mitochondrial apoptosis - PubMed
Description:
The multidomain proapoptotic molecules BAK or BAX are required to initiate the mitochondrial pathway of apoptosis. How cells maintain the potentially lethal proapoptotic effector BAK in a monomeric inactive conformation at mitochondria is unknown. In viable cells, we found BAK complexed with mitocho …
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {📚}

  • Science
  • Health & Fitness
  • Fitness & Wellness

Content Management System {📝}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,009,998 visitors per month in the current month.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {💸}

The income method remains a mystery to us.

Many websites are intended to earn money, but some serve to share ideas or build connections. Websites exist for all kinds of purposes. This might be one of them. Pubmed.ncbi.nlm.nih.gov might be making money, but it's not detectable how they're doing it.

Keywords {🔍}

protein, proteins, vdac, bak, doi, mitochondrial, pmid, apoptosis, bax, cells, voltagedependent, anion, metabolism, mitochondria, free, pmc, mouse, death, article, human, cited, channel, review, articles, mesh, bcl, cell, epub, channels, research, pharmacology, membrane, porins, protooncogene, main, states, ncbi, similar, resources, science, activation, proapoptotic, pathway, isoform, apoptotic, pubmed, release, cytochrome, biol, apr,

Topics {✒️}

united states government mitochondrial channel vdac main page content similar articles cited mouse bid protein human vdac1 protein mouse vdac2 protein mouse bax protein cell death differ funding linkout protein protein bcl-2 bcl human bak1 protein human vdac2 protein agrobacterium-mediated genetic transformation human bax protein monomeric inactive conformation books gene gene death signals activate mesh keyword pubchem substance grants mitochondrial apoptosis emily apoptotic death bak-mediated apoptosis abundant vdac1 bh3 cell metab mol cells mitochondrial apoptotic pathway enabling homo-oligomerization resources science vdac isoform present hhs 1083995 abstract medgen nucleotide omim research materials addgene core apoptotic pathway found bak complexed cells maintain viable cells cells deficient cells lacking cancer cells binding research support cited mitochondrial pathway bax activation biology

External Links {🔗}(102)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • jQuery

CDN Services {📦}

  • Ncbi

3.9s.