Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/12438411/.

Title:
Recombinant expression of the voltage-dependent anion channel enhances the transfer of Ca2+ microdomains to mitochondria - PubMed
Description:
Although the physiological relevance of mitochondrial Ca2+ homeostasis is widely accepted, no information is yet available on the molecular identity of the proteins involved in this process. Here we analyzed the role of the voltage-dependent anion channel (VDAC) of the outer mitochondrial membrane i …
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {πŸ“š}

  • Science
  • Fitness & Wellness
  • Education

Content Management System {πŸ“}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {πŸ’Έ}

We can't tell how the site generates income.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Pubmed.ncbi.nlm.nih.gov could be getting rich in stealth mode, or the way it's monetizing isn't detectable.

Keywords {πŸ”}

mitochondrial, doi, metabolism, pmid, pmc, biol, proteins, pubmed, voltagedependent, anion, vdac, cell, channel, cells, figure, channels, states, epub, mitochondria, apoptosis, chem, free, article, nih, articles, mesh, information, recombinant, homeostasis, skeletal, intracellular, review, endoplasmic, research, ion, protooncogene, hhsunited, main, ncbi, figures, similar, cited, references, resources, nov, jcb, expression, molecular, role, hela,

Topics {βœ’οΈ}

green fluorescent proteins united states government voltage-dependent anion channel endoplasmic reticulum-mitochondrial axis protein fura-2 recombinant expression skeletal muscle myotubes main page content proteins involved similar articles cited literature sources intact hela cells endoplasmic reticulum funding linkout hela cells ca2+ transport bcl-xl bcl-2 bcl mitochondrial ca2+ homeostasis articles references babcock agonist-dependent increases mesh keyword hhs resources molecular identity molecular component skeletal myotubes ceramide-induced cell death research support ca2+ homeostasis vdac-overexpressing cells mitochondria elena rapizziΒ 1 protein mitochondrial channel vdac 200205091 abstract mammalian cells high speed imaging cgi1746 targets Οƒ1r rat cortical astrocytes outer mitochondrial membrane ca2+-sensitive gfps ca2+ signaling pathways mitochondria exert initiating cell death curr med chem intracellular ca2+ network intracellular ca2+ waves curr mol med figures

External Links {πŸ”—}(95)

Analytics and Tracking {πŸ“Š}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {πŸ“š}

  • Clipboard.js
  • jQuery
  • PhotoSwipe

CDN Services {πŸ“¦}

  • Ncbi

3.95s.