Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/11782314/.

Title:
A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis - PubMed
Description:
The mechanism during apoptosis by which cytochrome c is rapidly and completely released in the absence of mitochondrial swelling is uncertain. Here, we show that two distinct pathways are involved. One mediates release of cytochrome c across the outer mitochondrial membrane, and another, characteriz …
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {πŸ“š}

  • Science
  • Fitness & Wellness
  • Education

Content Management System {πŸ“}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {πŸ’Έ}

We're unsure how the site profits.

Many websites are intended to earn money, but some serve to share ideas or build connections. Websites exist for all kinds of purposes. This might be one of them. Pubmed.ncbi.nlm.nih.gov might have a hidden revenue stream, but it's not something we can detect.

Keywords {πŸ”}

doi, proteins, cytochrome, pmid, mitochondrial, protein, free, article, pmc, metabolism, membrane, review, states, cell, cristae, release, protooncogene, nih, articles, cited, mesh, apoptosis, bak, mitochondria, cells, hhsunited, main, ncbi, similar, resources, distinct, korsmeyer, structure, domain, pubmed, proapoptotic, bid, bax, biol, death, bcl, caspase, jul, research, carrier, pharmacology, liver, pubchem, hhs, page,

Topics {βœ’οΈ}

united states government funding linkout main page content pro-apoptotic proteins similar articles cited hhs bh3 domain mouse bid protein cell death differ outer mitochondrial membrane mitochondrial outer membrane mitochondrial membrane lipids cancer stem cells pubchem substance grants mesh keyword resources dev cell bak- versus bax-dependence apoptosis luca scorranoΒ 1 permeability transition pore rat brain mitochondria de guevara rl mitochondria-cytoskeleton interactions intrinsic apoptosis molecule tbid induces bh3 resolving presynaptic structure cells bid-induced cytochrome bmc cell biol tbid-induced cytochrome cited apoptosis esposti md research support mitochondrial structure mitochondrial swelling pathway independent distinct roles electron mitochondria biomedical research cell biol require tbid' pubmed wordmark pubmed logo cell metab distinct pathways biol chem oligomerizes bak korsmeyer sj intramitochondrial cristae

External Links {πŸ”—}(69)

Analytics and Tracking {πŸ“Š}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {πŸ“š}

  • Clipboard.js
  • jQuery

CDN Services {πŸ“¦}

  • Ncbi

3.45s.