Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/11435345/.

Title:
Calcineurin plays a critical role in the development of pressure overload-induced cardiac hypertrophy - PubMed
Description:
These results suggest that calcineurin plays a critical role in the development of pressure overload-induced cardiac hypertrophy.
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {📚}

  • Health & Fitness
  • Science
  • Education

Content Management System {📝}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,058,662 visitors per month in the current month.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {💸}

We're unsure if the website is profiting.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Pubmed.ncbi.nlm.nih.gov might be earning cash quietly, but we haven't detected the monetization method.

Keywords {🔍}

calcineurin, doi, cardiac, hypertrophy, pmid, mice, pressure, role, overloadinduced, heart, transgenic, pmc, chen, review, free, article, articles, cir, weight, genes, main, ncbi, abstract, similar, cited, mesh, information, resources, circulation, plays, critical, yazaki, komuro, activation, wildtype, protein, kinases, pubmed, epub, res, molkentin, pathology, genetics, cardiomegaly, hhs, page, content, homepage, navigation, search,

Topics {✒️}

main page content pressure-overload cardiac hypertrophy blood pressure body weight activated t-cells pressure overload produced littermate wild-type mice early response genes pathologic disease models abdominal aorta cardiac calcineurin activity fetal-type genes dependent phosphatase calcineurin calcineurin-nuclear factor induce cardiomyocyte hypertrophy wild-type mice constriction similar articles cited united states government dorn gw 2nd renin-angiotensin system cardiac hypertrophy potential therapeutic target dominant negative mutant molecular mechanisms cholesterol-independent mechanisms resources circulation cell physiol biochem 2 t-type ca calcineurin plays transgenic mice dominant genes heart weight calcineurin specifically calcineurin inhibition dissect calcineurin' fibrosis abstract health hypertrophic responses calcineurin activation hhs hypertrophy pubmed wordmark pubmed logo official website present study significant differences basal state

Questions {❓}

  • Calcineurin and cardiac hypertrophy: where have we been?

External Links {🔗}(58)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • jQuery

CDN Services {📦}

  • Ncbi

3.38s.