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Topics {✒️}

sirt1/nf-kappab/b7-h3/tnf-alpha axis sirt1/nf‑κb/b7‑h3/tnf‑α axis [73] ccl2-ccr2-m2 macrophage axis jak1-stat3-sox2 signaling pathway b7h4-dependent anti-tumor immunity pi3k/akt/stat3 signaling pathway pi3k/akt/bcl-2 signaling pathway cell-dependent anti-tumor immunity low-dose doxorubicin-induced senescence tlr4/nf-κb pathway akt/tm4sf1/sirt1 pathway b7-h3 localization profiles fut8-mediated aberrant n-glycosylation crispr/cas9 mediated knockout anti-b7-h3 therapeutic agents b7-h3 post-translational modifications pi3k/akt/mtor signaling b7-h3 research b7-h3 neck squamous-cell carcinoma pi3k/akt signaling pathway immune receptors b7–h3/cd276 pi3k/akt/mtor pathway [91] pi3k/akt/mtor pathway mir‑34a induces immunosuppression mir-34a induces immunosuppression b-h3 inhibitors interferon-dependent ido induction b7-h3 targeted rit nk cell-mediated cytotoxicity full size image ifn-gamma-dependent cytotoxicity nf-κb pathway [79] nf-κb pathway nfat-dependent il-6 release pi3k/akt/mtor signalling b7-h3-induced signaling wnt/β-catenin pathway article download pdf anti-b7-h3 approaches special expression profile b7-h3 enhanced glycolysis successful targeting b7-h3 b7-h3 dramatically increased rhabdomyosarcoma reveals b7-h3 b7-h3 positively correlated abrogating jak2/stat3 phosphorylation mvp-mediated mek activation b7-h3 protein stability anti-tumor immune responses costimulatory human b7-h3

Questions {❓}

• The immune checkpoint inhibitors: where are we now?

Schema {🗺️}

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         headline:New frontiers in immune checkpoint B7-H3 (CD276) research and drug development
         description:B7-H3 (CD276), a member of the B7 family of proteins, is a key player in cancer progression. This immune checkpoint molecule is selectively expressed in both tumor cells and immune cells within the tumor microenvironment. In addition to its immune checkpoint function, B7-H3 has been linked to tumor cell proliferation, metastasis, and therapeutic resistance. Furthermore, its drastic difference in protein expression levels between normal and tumor tissues suggests that targeting B7-H3 with drugs would lead to cancer-specific toxicity, minimizing harm to healthy cells. These properties make B7-H3 a promising target for cancer therapy. Recently, important advances in B7-H3 research and drug development have been reported, and these new findings, including its involvement in cellular metabolic reprograming, cancer stem cell enrichment, senescence and obesity, have expanded our knowledge and understanding of this molecule, which is important in guiding future strategies for targeting B7-H3. In this review, we briefly discuss the biology and function of B7-H3 in cancer development. We emphasize more on the latest findings and their underlying mechanisms to reflect the new advances in B7-H3 research. In addition, we discuss the new improvements of B-H3 inhibitors in cancer drug development.
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      headline:New frontiers in immune checkpoint B7-H3 (CD276) research and drug development
      description:B7-H3 (CD276), a member of the B7 family of proteins, is a key player in cancer progression. This immune checkpoint molecule is selectively expressed in both tumor cells and immune cells within the tumor microenvironment. In addition to its immune checkpoint function, B7-H3 has been linked to tumor cell proliferation, metastasis, and therapeutic resistance. Furthermore, its drastic difference in protein expression levels between normal and tumor tissues suggests that targeting B7-H3 with drugs would lead to cancer-specific toxicity, minimizing harm to healthy cells. These properties make B7-H3 a promising target for cancer therapy. Recently, important advances in B7-H3 research and drug development have been reported, and these new findings, including its involvement in cellular metabolic reprograming, cancer stem cell enrichment, senescence and obesity, have expanded our knowledge and understanding of this molecule, which is important in guiding future strategies for targeting B7-H3. In this review, we briefly discuss the biology and function of B7-H3 in cancer development. We emphasize more on the latest findings and their underlying mechanisms to reflect the new advances in B7-H3 research. In addition, we discuss the new improvements of B-H3 inhibitors in cancer drug development.
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      name:Cancer Research Institute and School of Basic Medical Sciences, Central South University, Changsha, China
      name:Department of Biochemistry and Molecular Biology, College of Medicine, University of Florida, Gainesville, USA
      name:Department of Tumor Biology, Institute for Cancer Research, Oslo University Hospital Radiumhospitalet, Oslo, Norway
      name:Institute of Biochemistry and Molecular Biology, Institute of Biomedical Sciences, and Research Center for Cancer Biology, China Medical University, Taichung, Taiwan

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