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LINK . SPRINGER . COM {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Link.springer.com Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. CDN Services

We are analyzing https://link.springer.com/article/10.1186/bcr578.

Title:
Host microenvironment in breast cancer development: Epithelial–mesenchymal transition in breast cancer development | Breast Cancer Research
Description:
The epithelial–mesenchymal transition (EMT) is a developmental mechanism of crucial importance in establishing the body plan in many multicellular organisms. Several transduction pathways controlling the various steps of the morphological transition have been identified by molecular analyses of this process in cell lines and in vivo. The newly formed mesenchymal cells can exhibit locomotory and invasive phenotypes, suggesting that EMTs contribute to the progression of carcinoma. Diverse evidence indicates that EMT subprograms are involved in the appearance of different breast carcinoma types. Several normal and malignant breast cell lines are currently being analyzed to define key steps in EMT and to identify candidate genes. DNA profiling technology is also being applied to uncover pathways that lead to a metastatic phenotype.
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {📚}

  • Education
  • Science
  • Telecommunications

Content Management System {📝}

What CMS is link.springer.com built with?

Custom-built

No common CMS systems were detected on Link.springer.com, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of link.springer.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Link.springer.com Make Money? {💸}

We find it hard to spot revenue streams.

While many websites aim to make money, others are created to share knowledge or showcase creativity. People build websites for various reasons. This could be one of them. Link.springer.com might be cashing in, but we can't detect the method they're using.

Keywords {🔍}

breast, cell, pubmed, google, scholar, article, cells, emt, cas, carcinoma, cancer, epithelial, ecadherin, tumor, carcinomas, tumors, mammary, development, snail, growth, lines, gene, expression, biol, transition, invasive, factors, central, thiery, gland, epithelialmesenchymal, pathways, vitro, mesenchymal, genes, differentiation, transcription, found, progression, involved, phenotype, mechanisms, line, tgfβ, type, large, composed, van, types, related,

Topics {✒️}

jean paul thiery ras/mitogen-activated protein kinase article vincent-salomon specifically high-molecular-mass cytokeratins zinc-finger transcription factors e-cadherin wild-type locus transforming growth factor-beta transforming growth factor-β n-cadherin de novo repressing e-cadherin expression epithelial–mesenchymal transition //breast-cancer-research epithelial growth factor tgf-β-mediated induction tgf-β induced transdifferentiation vincent-salomon epidermal growth factor e-cadherin gene extinction epithelial–mesenchymal transitions undergoing extensive migration epithelial-mesenchymal transformations fibroblast growth factors privacy choices/manage cookies van rooijen ma nat rev cancer dna profiling technology mediates cell scattering tgf-β signaling favorable clinical outcome cancer metastasis rev epithelio-mesenchymal transformation perez-moreno ma author correspondence human e-cadherin long-term follow epithelial cell plasticity epithelial cell lines epithelial cell intercalation find studies differing skin carcinogenesis model murine hc11 clones institut curie rqs=heppner rights human breast carcinoma breast cancer micrometastases implicating e-cadherin e-cadherin downregulation e-cadherin complex building epithelial architecture tumor growth rate

Questions {❓}

  • Ilyas M: Adhesion molecule expression in breast cancer: the phoenix in tumour metastasis?

Schema {🗺️}

WebPage:
      mainEntity:
         headline:Host microenvironment in breast cancer development: Epithelial–mesenchymal transition in breast cancer development
         description:The epithelial–mesenchymal transition (EMT) is a developmental mechanism of crucial importance in establishing the body plan in many multicellular organisms. Several transduction pathways controlling the various steps of the morphological transition have been identified by molecular analyses of this process in cell lines and in vivo. The newly formed mesenchymal cells can exhibit locomotory and invasive phenotypes, suggesting that EMTs contribute to the progression of carcinoma. Diverse evidence indicates that EMT subprograms are involved in the appearance of different breast carcinoma types. Several normal and malignant breast cell lines are currently being analyzed to define key steps in EMT and to identify candidate genes. DNA profiling technology is also being applied to uncover pathways that lead to a metastatic phenotype.
         datePublished:2003-04-01T00:00:00Z
         dateModified:2003-04-01T00:00:00Z
         pageStart:1
         pageEnd:6
         sameAs:https://doi.org/10.1186/bcr578
         keywords:
            breast carcinoma
            E-cadherin
            epithelial-mesenchymal transition
            growth factors
            Snail
            Cancer Research
            Oncology
            Surgical Oncology
         image:
         isPartOf:
            name:Breast Cancer Research
            issn:
               1465-542X
            volumeNumber:5
            type:
               Periodical
               PublicationVolume
         publisher:
            name:BioMed Central
            logo:
               url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
               type:ImageObject
            type:Organization
         author:
               name:Anne Vincent-Salomon
               affiliation:
                     name:Institut Curie
                     address:
                        name:Medical Division, Department of Tumor Biology, Institut Curie, Paris, France
                        type:PostalAddress
                     type:Organization
               type:Person
               name:Jean Paul Thiery
               affiliation:
                     name:UMR 144 CNRS Institut Curie
                     address:
                        name:UMR 144 CNRS Institut Curie, Paris, France
                        type:PostalAddress
                     type:Organization
               email:[email protected]
               type:Person
         isAccessibleForFree:1
         type:ScholarlyArticle
      context:https://schema.org
ScholarlyArticle:
      headline:Host microenvironment in breast cancer development: Epithelial–mesenchymal transition in breast cancer development
      description:The epithelial–mesenchymal transition (EMT) is a developmental mechanism of crucial importance in establishing the body plan in many multicellular organisms. Several transduction pathways controlling the various steps of the morphological transition have been identified by molecular analyses of this process in cell lines and in vivo. The newly formed mesenchymal cells can exhibit locomotory and invasive phenotypes, suggesting that EMTs contribute to the progression of carcinoma. Diverse evidence indicates that EMT subprograms are involved in the appearance of different breast carcinoma types. Several normal and malignant breast cell lines are currently being analyzed to define key steps in EMT and to identify candidate genes. DNA profiling technology is also being applied to uncover pathways that lead to a metastatic phenotype.
      datePublished:2003-04-01T00:00:00Z
      dateModified:2003-04-01T00:00:00Z
      pageStart:1
      pageEnd:6
      sameAs:https://doi.org/10.1186/bcr578
      keywords:
         breast carcinoma
         E-cadherin
         epithelial-mesenchymal transition
         growth factors
         Snail
         Cancer Research
         Oncology
         Surgical Oncology
      image:
      isPartOf:
         name:Breast Cancer Research
         issn:
            1465-542X
         volumeNumber:5
         type:
            Periodical
            PublicationVolume
      publisher:
         name:BioMed Central
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Anne Vincent-Salomon
            affiliation:
                  name:Institut Curie
                  address:
                     name:Medical Division, Department of Tumor Biology, Institut Curie, Paris, France
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Jean Paul Thiery
            affiliation:
                  name:UMR 144 CNRS Institut Curie
                  address:
                     name:UMR 144 CNRS Institut Curie, Paris, France
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
      isAccessibleForFree:1
["Periodical","PublicationVolume"]:
      name:Breast Cancer Research
      issn:
         1465-542X
      volumeNumber:5
Organization:
      name:BioMed Central
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:Institut Curie
      address:
         name:Medical Division, Department of Tumor Biology, Institut Curie, Paris, France
         type:PostalAddress
      name:UMR 144 CNRS Institut Curie
      address:
         name:UMR 144 CNRS Institut Curie, Paris, France
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:Anne Vincent-Salomon
      affiliation:
            name:Institut Curie
            address:
               name:Medical Division, Department of Tumor Biology, Institut Curie, Paris, France
               type:PostalAddress
            type:Organization
      name:Jean Paul Thiery
      affiliation:
            name:UMR 144 CNRS Institut Curie
            address:
               name:UMR 144 CNRS Institut Curie, Paris, France
               type:PostalAddress
            type:Organization
      email:[email protected]
PostalAddress:
      name:Medical Division, Department of Tumor Biology, Institut Curie, Paris, France
      name:UMR 144 CNRS Institut Curie, Paris, France

External Links {🔗}(172)

Analytics and Tracking {📊}

  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • Prism.js

CDN Services {📦}

  • Crossref

4.7s.