We are analyzing https://link.springer.com/article/10.1186/1475-2840-8-23.

Title:
Postprandial hyperglycemia as an etiological factor in vascular failure | Cardiovascular Diabetology
Description:
Postprandial hyperglycemia is characterized by hyperglycemic spikes that induce endothelial dysfunction, inflammatory reactions and oxidative stress, which may lead to progression of atherosclerosis and occurrence of cardiovascular events. Emerging data indicate that postprandial hyperglycemia or even impaired glucose tolerance may predispose to progression of atherosclerosis and cardiovascular events. There is evidence that postprandial hyperglycemia, but not fasting hyperglycemia, independently predicts the occurrence of cardiovascular events. We proposed a concept of
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Topics {✒️}

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Questions {❓}

DECODE Study Group: Is the current definition of diabetes relevant to mortality risk from all causes and cardiovascular and noncardiovascular diseases?

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WebPage:
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         headline:Postprandial hyperglycemia as an etiological factor in vascular failure
         description:Postprandial hyperglycemia is characterized by hyperglycemic spikes that induce endothelial dysfunction, inflammatory reactions and oxidative stress, which may lead to progression of atherosclerosis and occurrence of cardiovascular events. Emerging data indicate that postprandial hyperglycemia or even impaired glucose tolerance may predispose to progression of atherosclerosis and cardiovascular events. There is evidence that postprandial hyperglycemia, but not fasting hyperglycemia, independently predicts the occurrence of cardiovascular events. We proposed a concept of 'vascular failure' as a comprehensive syndrome of vascular dysfunction extending from risk factors to advanced atherosclerotic disease. Postprandial hyperglycemia is therefore one of the very important pathophysiological states contributing to vascular failure. Accordingly, controlling postprandial hyperglycemia should be the focus of future clinical investigation as a potential target for preventing vascular failure.
         datePublished:2009-04-29T00:00:00Z
         dateModified:2009-04-29T00:00:00Z
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            Impair Glucose Tolerance
            Pioglitazone
            Acarbose
            Exenatide
            Diabetes
            Angiology
            Cardiology
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ScholarlyArticle:
      headline:Postprandial hyperglycemia as an etiological factor in vascular failure
      description:Postprandial hyperglycemia is characterized by hyperglycemic spikes that induce endothelial dysfunction, inflammatory reactions and oxidative stress, which may lead to progression of atherosclerosis and occurrence of cardiovascular events. Emerging data indicate that postprandial hyperglycemia or even impaired glucose tolerance may predispose to progression of atherosclerosis and cardiovascular events. There is evidence that postprandial hyperglycemia, but not fasting hyperglycemia, independently predicts the occurrence of cardiovascular events. We proposed a concept of 'vascular failure' as a comprehensive syndrome of vascular dysfunction extending from risk factors to advanced atherosclerotic disease. Postprandial hyperglycemia is therefore one of the very important pathophysiological states contributing to vascular failure. Accordingly, controlling postprandial hyperglycemia should be the focus of future clinical investigation as a potential target for preventing vascular failure.
      datePublished:2009-04-29T00:00:00Z
      dateModified:2009-04-29T00:00:00Z
      pageStart:1
      pageEnd:10
      license:http://creativecommons.org/licenses/by/2.0
      sameAs:https://doi.org/10.1186/1475-2840-8-23
      keywords:
         Endothelial Dysfunction
         Impair Glucose Tolerance
         Pioglitazone
         Acarbose
         Exenatide
         Diabetes
         Angiology
         Cardiology
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